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Titlebook: Cardiac Adaptation to Hemodynamic Overload, Training and Stress; R. Jacob,R. W. Gülch,G. Kissling Conference proceedings 1983 Dr. Dietrich

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书目名称Cardiac Adaptation to Hemodynamic Overload, Training and Stress
编辑R. Jacob,R. W. Gülch,G. Kissling
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图书封面Titlebook: Cardiac Adaptation to Hemodynamic Overload, Training and Stress;  R. Jacob,R. W. Gülch,G. Kissling Conference proceedings 1983 Dr. Dietrich
描述Cardiac hypertrophy and accompanying phenomena have received increasing attention in recent years - particularly in the basic sciences. The present volume contains the proceedings of the Erwin RIESCH SYMPOSIUM on "CARDIAC ADAPTATION TO HEMODYNAMIC OVERLOAD, TRAINING AND STRESS" held in Tiibingen on Sep­ tember 19-22, 1982. In addition to the topics of the previous symposia (1976 and 1979) concerned with problems of cardiac hypertrophy, the scope of this sequel meeting has been expanded to include related fields. The intention was to consider numerous related features and problems of chronic reactions of the heart (and vascular system) to abnormal hemodynamic loading, as well as alterations due to maturation, aging, training, neuroendocrine status and stress. Special attention has been paid to cardiac reactions at the level of contractile proteins. The results are considered primarily in light of long-term adaptation of the heart. Of course, research at the forefront of current knowledge need not always lead to congruent conclusions. Neither can the individual contributions always agree with the viewpoint of the editors. However, the broad array of individual approaches employed by
出版日期Conference proceedings 1983
关键词adaptation; aging; artery; attention; blood pressure; cardiovascular; circulation; heart; heart disease; hype
版次1
doihttps://doi.org/10.1007/978-3-642-85326-5
isbn_softcover978-3-642-85328-9
isbn_ebook978-3-642-85326-5
copyrightDr. Dietrich Steinkopff Verlag, GmbH & Co. KG, Darmstadt 1983
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Relationship between the synthesis rates and mRNA levels of the α and β cardiac myosin heavy chains ssion of myosin in the ventricles in both the euthyroid and thyrotoxic states..Administration of D-thyroxine, like the naturally occurring isomer, also resulted in a relative accumulation of HCa in the ventricles.
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Cardiac Adaptation to Hemodynamic Overload, Training and Stress
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Cardiac Adaptation to Hemodynamic Overload, Training and Stress978-3-642-85326-5
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https://doi.org/10.1007/0-387-32995-1es at the myofibrillar level which can be attributed solely to changes in mechanical load..With regard to human myocardium, further investigation will be required to determine the extent of possible changes in functional parameters attributable to variation of the myosin molecule, including the ligh
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