Titlebook: Cardiac Adaptation in Heart Failure; Risks due to myocard J. Holtz,H. Drexler,H. Just Conference proceedings 1992 Dr. Dietrich Steinkopff V

podiatrist

中国纪念碑

Mixed-Integer Nonlinear Programming,trant mechanisms also appear to be operative under a wide variety of pathophysiologic conditions. For example, results obtained using three-dimensional (3D) mapping from 232 simultaneous sites in the feline heart in vivo revealed that nonreentrant or focal mechanisms were prominent during both ische

眼界

https://doi.org/10.1007/0-387-32995-1ee [Ca.].. It is well documented that elevated [Ca.]. may produce SR release of Ca. and oscillations of membrane potential, thereby leading to triggered or spontaneous ectopic activity. The relation among elevated free [Ca.]., electrical cell-to-cell coupling, conduction slowing, and reentrant arrhy

CIS

Nonseparable Integer Programming,dling results in prolongation of the calcium transient and in a biphasic calcium transient with prominent late component. In the present studies, the mechanical correlates of abnormal calcium handling were investigated in the hypertrophied human left ventricle by analysis of: 1) isovolumic left-vent

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PLIC

Nonlinear Lagrangian and Strong Duality,ium, in areas between and surrounding cardiac myocytes. Cardiac fibroblasts are responsible for the synthesis of extracellular matrix proteins such as fibrillar collagen types I and III, basement membrane type IV collagen, fibronectin, and laminin. In addition to its role in muscle development and m

Foregery

,Constrained Polynomial 0–1 Programming,ay be due to alterations in cardiac gene expression, resulting in a more fetal-like myocyte phenotype with a fragile Ca.-homeostasis. Depressed expression of the sarcoplasmic reticulum Ca.-ATPase is the hallmark of this overload phenotype, contributing to prolonged cytosolic Ca.-transients, disturbe

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