Titlebook: Cancer and Inflammation; Douglas W. Morgan,Ulf J. Forssmann,Marian T. Nakad Book 2004 Birkhäuser Verlag 2004 Chemokine.angiogenesis.cancer

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Johanna Ennser-Kananen,Taina Saarinenmal tissue integrity. Both show evidence of dysregulated proliferation of particular cell types and consequently both are associated with hyperplasias. In addition, both chronic inflammation and cancer are also dependent on subverting the process of programmed cell death. The term “apoptosis” was fi

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Johanna Ennser-Kananen,Taina Saarineners of the gastrointestinal tract were responsible for more than 40% of all cancer deaths, nearly 2.2 million people [., .]. Interestingly, the incidence of these cancers can vary considerably between regions. This is most likely due to a variety of factors including environmental exposures, diet, s

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Johanna Ennser-Kananen,Taina SaarinenMMPs are a family of at least 25 highly homologous secreted or plasma membrane-associated zinc-binding proteinases (reviewed in [.,.]). MMPs can be produced by virtually all cell types, and regulate many developmental processes and a variety of pathological conditions, including, but not limited to, cancer, auto-immune diseases, and periodontitis.

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Discontinuous Coatings — e.g. by Printingwith many of the therapeutic targets seemingly intractable. What are we missing? The editors of this volume have addressed the relatedness of these seemingly “unrelated” fields to reveal “meaningfulness” that is now more perceptible. This revelation could provide a synergistic base to allow the gene

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1422-7746 rates on clinical studies with antiinflammatory drugs in cancer treatment. The volume is written for biomedical researchers in the health care industry and in academia who are working in these areas..978-3-0348-9600-9978-3-0348-7861-6Series ISSN 1422-7746 Series E-ISSN 2296-4525