Titlebook: Cancer Stem Cell Markers and Related Network Pathways; Shihori Tanabe Book 2022 The Editor(s) (if applicable) and The Author(s), under exc

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0065-2598 book entitled “Cancer Stem Cell Markers and Related Network Path-ways” is about cancer stem cell (CSC) markers and the molecular network pathways. CSCs play an important role in the cancer drug resistance, metastasis and recurrence. Epithelial-mesenchymal transition (EMT) is closely related to CSC p

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Blue/UV-A Light Signaling in Moss,rkers of CSCs have been correlated with maintaining survival, self-renewal and extravasation properties. Here, we highlight the importance of glycosylation in promoting the stemness character of CSCs and the current strategies for targeting abnormal glycosylation toward generating effective therapies against the CSC population.

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Abnormal Glycosylation in Cancer Cells and Cancer Stem Cells as a Therapeutic Target,rkers of CSCs have been correlated with maintaining survival, self-renewal and extravasation properties. Here, we highlight the importance of glycosylation in promoting the stemness character of CSCs and the current strategies for targeting abnormal glycosylation toward generating effective therapies against the CSC population.

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ERBB Signaling Pathway in Cancer Stem Cells, CSC characters such as survival, tumorigenicity and stemness. Because of the roles in tumor growth and progress, ERBBs are considered to be one of the drug targets as cancer treatment strategy. In this chapter, we will summarize the structure, function and roles of ERBB subfamily along with their r

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Luisa Anderloni,Emanuele Maria Carluccio CSC characters such as survival, tumorigenicity and stemness. Because of the roles in tumor growth and progress, ERBBs are considered to be one of the drug targets as cancer treatment strategy. In this chapter, we will summarize the structure, function and roles of ERBB subfamily along with their r

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Cancer Stem Cell Markers and Related Network Pathways

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https://doi.org/10.1007/978-3-031-12974-2Epithelial-mesenchymal transition; Gene expression; Cellular signaling; Drug resistance; Cancer therapy