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Titlebook: Calcium in Drug Actions; Peter F. Baker (Professor Sc. D., F.R.S.) Book 1988 Springer-Verlag Berlin Heidelberg 1988 ATP.ATPase.Calcium.bon

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https://doi.org/10.1007/978-3-642-46961-9he original report of control and prevention of mania by Li. salts (C. 1949) initiated widespread use of Li. for this illness. Since Li. is most effective in reducing the mood swings of bipolar manic-depressive patients, patients suffering from this disease often take Li. for the rest of their lives
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,Die neue Sicht — Anstöße und Begründungen,d clinical investigations for some time, and some of them have been developed into useful therapeutic drugs for the treatment of hypertension and coronary disorders (F. 1977; . 1981). However, most of these studies were performed with classical pharmacologic techniques involving measurement of muscl
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W. Heller,D. Hoffmann,H. Junger,H. Vontinery few processes that the precise location of the calcium-sensitive effector has been identified. Two examples stand out. In skeletal muscle it is known beyond doubt that calcium interacts with troponin C, a satellite protein of the actomyosin, and through the consequent activation of myosin ATPase
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https://doi.org/10.1007/978-3-662-25270-3philic secretion products (e.g., proteins, peptides, classical neurotransmitters, such as amines, acetylcholine, and amino acids) and to discharge these products by a process known as exocytosis. The term exocytosis was originally proposed to describe the release in bulk of the contents of a membran
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https://doi.org/10.1007/978-3-642-71806-9ATP; ATPase; Calcium; bone; lead; metabolism; proteins; smooth muscle
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978-3-642-71808-3Springer-Verlag Berlin Heidelberg 1988
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Calcium in Drug Actions978-3-642-71806-9Series ISSN 0171-2004 Series E-ISSN 1865-0325
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