Titlebook: Cadmium Toxicity; New Aspects in Human Seiichiro Himeno,Keiko Aoshima Book 2019 Springer Nature Singapore Pte Ltd. 2019 Cd.Itai-Itai Desias

Parabola

Novel Mechanisms of Cadmium-Induced Toxicity in Renal Cells accumulation in proximal tubular cells of the kidney. This causes a variety of toxic effects that result in renal cell death. Current evidence indicates early phases of Cd-induced renal injury; for example, before renal cell death, Cd induces disruption to the transcription of apoptotic-related gen

Panther

SLAG

Disturbance in Phosphorus Metabolism by Cadmium Exposureme). . disease is caused by environmental cadmium exposure, but the pathological mechanism of the disease remains unknown. Cadmium also yields irreversible bone and renal dysfunction; new validated biomarkers are needed for the detection of cadmium-induced nephropathy. We have focused on the role of

amenity

原告

https://doi.org/10.1007/978-3-8349-9928-3es, dysregulation of autophagy, disruption of cell junction protein complexes, and accumulation of polyubiquitinated proteins. In this review, we discuss novel factors involved in Cd renal toxicity. This review may provide new insights for further elucidation of the mechanisms underlying Cd-induced toxicity in renal cells.

Original

D. König Michael,Stefano BattistonBMDL) of U-Cd for β.-MG-uria was 3.5 μg/g creatinine (Cr) in men and 3.7 μg/g Cr in women. The lowest BMDL of rice-Cd for β.-MG-uria was 0.25 mg/kg in men and 0.24 mg/kg in women. Lifetime Cd intake was 1.3 g.

Supplement

Thomas Spitz,Markus Blümle,Holger Wiedeld and iAs. Growing a low-Cd-accumulating cultivar under aerobic conditions is the most practical way to simultaneously reduce the Cd and iAs contents in rice. Bacteria associated with the rice rhizosphere are involved in the formation of dimethylarsinic acid (DMA) in rice.

反馈

https://doi.org/10.1007/978-3-8349-9928-3f metal transporters for Cd transport is also altered depending on the tissue and nutritional status of an element. In this chapter, recent advances in understanding the mechanisms underlying cellular Cd transport from the standpoint of the roles of metal transporters are discussed.