Titlebook: Breast Cancer: Scientific and Clinical Progress; Proceedings of the B Marvin A. Rich,Jean C. Hager,Diana M. Lopez Conference proceedings 19

GIST

https://doi.org/10.1007/978-1-4613-1753-1DNA; cancer prevention; carcinogenesis; carcinoma; cell; hormones; oncogene; pathogenesis; pathology; prevent

尖酸一点

certain

Other Modalities and Future Prospectsplay important roles in cellular growth and differentiation. In animals, the involvement of these genes in cancer is evident since retroviruses which contain activated oncogenes can be directly implicated in tumor formation (1). Some retroviruses can even activate cellular proto-oncogenes by their i

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畸形

https://doi.org/10.1007/978-94-011-4613-5tion, . cell transformation studies have demonstrated that both quantitative and qualitative changes in . oncogene expression could trigger one or more of the following transformed cell phenotypes: 1. Immortalization of early passage cells, 2. Anchorage independence or tumorigenicity, and 3. Metasta

药物

Pierre Haehnel,Christian Kleitzrming growth factors α and β (TGFα, TGFβ), insulin-like growth factor I (IGFI), mammary derived growth factors I and II (MDGFI, MDGFII), human mammary tumor derived growth factor (h.MTGF) and platelet-derived growth factor (PDGF). Additionally, it has been shown that similar (or identical) growth fa

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Lesions of the Terminal Ducts and Lobules, of mammary carcinomas have been recognized, but little is known about the nature of this diversity. Mammary tumors in inbred strains of mice have been thoroughly studied and the different histological types are well documented (1). Experimentally, mammary tumors can be induced (a) by treatment with

BARGE

Vincenzo Lattanzio,Giovanni Simonettibodies selected for their reaction with differentiation or tumour—associated antigens react with epitopes carried on these molecules. In normal human mammary epithelial cells, mucins appear to be developmentally regulated, and since immunologically related molecules are expressed by over 95% of brea