Titlebook: Breast Cancer Chemosensitivity; Dihua Yu,Mien-Chie Hung Book 2007 The Editor(s) (if applicable) and The Author(s), under exclusive license

小费

书目名称Breast Cancer Chemosensitivity影响因子(影响力)




书目名称Breast Cancer Chemosensitivity影响因子(影响力)学科排名




书目名称Breast Cancer Chemosensitivity网络公开度




书目名称Breast Cancer Chemosensitivity网络公开度学科排名




书目名称Breast Cancer Chemosensitivity被引频次




书目名称Breast Cancer Chemosensitivity被引频次学科排名




书目名称Breast Cancer Chemosensitivity年度引用




书目名称Breast Cancer Chemosensitivity年度引用学科排名




书目名称Breast Cancer Chemosensitivity读者反馈




书目名称Breast Cancer Chemosensitivity读者反馈学科排名

travail

语言学

Roles of Multidrug Resistance Genes in Breast Cancer Chemoresistance,wever, attempts to modulate the activities of these transporters using reversal agents have met with limited success. Future studies should focus on better understanding of the upregulation mechanisms of ABC transporter genes in breast cancers, and of the pharmacologic mechanisms of transporter-reve

sterilization

Therapy-Induced Apoptosis in Primary Tumors,models, the evidence that apoptosis does or does not mediate the effects of current front line therapies in patients, and the new strategies that are emerging that are designed to more directly target apoptotic pathways.

Charade

,Cell Cycle Deregulation in Breast Cancer: Insurmountable Chemoresistance or Achilles’ Heel?,tive measure of response..This review describes the mechanisms of resistance to commonly used systemic therapies for the treatment of breast cancer, with particular respect to the role of the cell cycle. The mechanisms and effects of the deregulation of cyclin E in breast cancer are reviewed and nov

negotiable

Integrin-Mediated Adhesion: Tipping the Balance between Chemosensitivity and Chemoresistance,ecent evidence suggests that integrin-mediated adhesion to the ECM may undermine the response of tumors to chemotherapeutic agents. Integrins have been shown to be readily accessible drug targets and are therefore attractive potential targets for combined modality chemotherapy.

ambivalence

Novel Approaches for Chemosensitization of Breast Cancer Cells: The E1A Story,the activity of a critical survival factor Akt and/or upregulating the activities of a pro-apoptotic kinase p38 and a protein phosphatase PP2A, etc. This review summarizes these progresses and proposes a plausible feed-forward model for E1A-mediated chemosensitization in human breast cancer cells.

Erythropoietin

obstruct

Public Sphere in the Digital Age,wever, attempts to modulate the activities of these transporters using reversal agents have met with limited success. Future studies should focus on better understanding of the upregulation mechanisms of ABC transporter genes in breast cancers, and of the pharmacologic mechanisms of transporter-reve

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