Titlebook: Breast Cancer Biology for the Radiation Oncologist; Jonathan Strauss,William Small,Gayle E. Woloschak Book 2015 Springer-Verlag Berlin Hei

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A Volatile Mix of Business and Politicsolymorphisms (SNPs) in many genes have been correlated to a higher risk of acute or late radiation toxicity. This chapter discusses cellular studies predictive of radiation sensitivity and studies of specific genes of interest, with a focus on breast cancer research. Radiogenomics studies of ATM, XR

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The Destruction of a Designated Heirely recent identification of the BRCA1 and BRCA2 genes in the mid-1990s, the expense associated with testing, and the inherent selection biases, the available literature has inherent limitations with relatively small patient numbers and lack of prospective randomized trials in this subset of patient

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From Outcast to Presidential Premiertherapy is designed to maximize therapeutic potential while minimizing harm to surrounding healthy tissue. Recently, advances in material design, synthesis, and characterization have lead to a surge in the number of approaches intended to shrink or eliminate the tumor burden of breast cancer. These

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Breast Cancer Biology for the Radiation Oncologist978-3-642-31220-5Series ISSN 0942-5373 Series E-ISSN 2197-4187

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https://doi.org/10.1007/978-981-15-2833-0r pathways responsible for regulating cellular proliferation, apoptosis, and genome integrity that are altered in DCIS. We find surprisingly few differences on a genomic or gene-expression level between DCIS and invasive disease. We introduce diverse models of DCIS progression that are stochastic ve

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https://doi.org/10.1007/978-981-15-2833-0 as the HER2-NFκB-HER2 signaling loop in the radioresistant BCSCs will be illustrated. Further clarification of the specific networks associated with the radioresistant phenotype of BCSCs will shed new light on the molecular mechanism of tumor radioresistance, and will help to generate targets to de

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