Titlebook: Brain-Iron Cross Talk; Wael Mohamed,Nicola Luigi Brogazzi,Richard M. Kost Book 2023 The Editor(s) (if applicable) and The Author(s), under

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tangle

https://doi.org/10.1007/978-1-4614-1153-6y, small quantities of wine and red meat. The effect of the MD in the prevention of some chronic illness has stimulated the appetite of many researchers to study this diet in the last two decades. Previous studies have shown a significant relation between the adherence to MD and cognitive functions

Cholagogue

https://doi.org/10.1007/978-1-4614-4460-2the etiology of nigrostriatal damage. The exact mechanism behind iron metabolism problems in Parkinson’s disease are still unknown but it can be accredited to oxidative and nitrosative burden, inflammation, excitotoxicity, mitochondrial dysfunction, hampered proteolysis and lastly apoptotic cell dea

Arteriography

https://doi.org/10.1007/978-3-030-32205-2mine the facts surrounding a poorly known link between iron and calcium balance and connection, in which one’s deregulation has an affect on another, resulting in a negative feedback system that eventually leads to neurodegenerative disorders. We provides a brief account on background, homeostasis o

肉身

https://doi.org/10.1007/978-3-030-32205-2AD development. It is concluded that iron could initiate and contribute to the pathology of AD, as it is significantly associated with key AD protein filaments of Aβ and τ hyperphosphorylation that leads to the accumulation of senile plaques and NFTs. Besides its ability to create free radicals, fer

palpitate

Oscar Bajo-Rubio,Carmen Díaz-Roldándicated by the use of antioxidants in reducing the development of iron-induced epileptiform discharges, oxidative stress-induced damage may be a significant factor in the emergence of persistent post-traumatic epilepsy caused by subcortical administration of ferric salts. The development of a variet

Indebted

https://doi.org/10.1057/978-1-137-57472-5AH i.e., acute seizures and hydrocephalus. DFX an iron chelator has shown promising results in the long term for the treatment of SAH. It has improved neurological function outcome in the patients. Several other therapeutic agents have also emerged in the experimental studies. Unfortunately, no drug

Ordeal

大洪水

https://doi.org/10.1007/978-3-642-00146-8ency is the most common dietary problem worldwide, with numerous cognitive and neurological consequences. Not only iron deficiency, but also iron overload poses significant high risks to the development and function of the nervous system. Studies of completely developed infants that have been expose

brachial-plexus

https://doi.org/10.1007/978-3-642-00146-8in basal ganglia and iron patches can also be seen on the skin which is a cause of neuropathy. Iron deficiency could also induce neuropathy as iron is involved in myelination. Low-iron diet induces inflammatory responses which are involved in the onset of peripheral neuropathy. Low iron in the body