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Titlebook: Brain and Heart Infarct; Proceedings of the T K. J. Zülch,W. Kaufmann (Direktor),V. Hossmann Conference proceedings 1977 Springer-Verlag Be

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楼主: Coarse
发表于 2025-4-1 03:54:23 | 显示全部楼层
Vascular Reactivity and Ion Homeostasis in Heart and Brain,he last years on calcium antagonistic substances. In cerebral vascular disorders the main interest until recently was not placed on Ca. but on the actions of H. and K. ions. In numerous metabolically linked cerebral circulatory disturbances it was demonstrated that the concentrations of these ions d
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Common Denominators in the Regulation of Blood Flow in Heart and Brain,oncerning the relative responses to specific factors that influence blood flow in each tissue. Some of these differences will be considered but the primary focus of this communication will be on the similarities of the control of cerebral and myocardial blood flow.
发表于 2025-4-1 13:14:34 | 显示全部楼层
Oxygen Transport in Heart and Brain,d carbon dioxide increases from the arterial to the venous end of the capillary. There is not a single oxygen tension in the tissue but an oxygen tension field: within the Krogh cylinder oxygen decreases both from the arterial to the venous end of the capillary, and perpendicularly from the capillar
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Regional Metabolism of the Brain,changes in regional function. Much inferential evidence links the metabolic rate and blood flow of a region of the brain to its level of functional activity. In fact, speculation concerning the link between metabolism, flow and function goes back to the work of ROY and SHERRINGTON in 1890 who postul
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Postischemic Amelioration of Brain Damage, that postischemic (PI) pathophysiological and biochemical changes . by the insult, . to the ultimate degree of neurologic deficit sustained. Also, that these PI pathologic changes can be prevented or ameliorated by early application of effective therapy.
发表于 2025-4-2 05:42:07 | 显示全部楼层
Coagulation Disturbances in Cerebrovascular Disorders,elopment of atherosclerosis (18, 23, 26, 30); and secondly thrombi are usually formed on an atherosclero-tically damaged vessel wall giving rise to severe stenosis or even occlusion of the artery which results finally in infarction (18, 30).
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