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Titlebook: Brain Tumor Immunotherapy; Linda M. Liau,Donald P. Becker,Darell D. Bigner Book 2001 Springer Science+Business Media New York 2001 apoptos

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https://doi.org/10.1007/978-3-031-43412-9when conjugated to a strong protein carrier, such as keyhole limpet hemocyanin (KLH), G. stimulates only a weak IgG response. It is also well known that shed gangliosides (e.g., G.) can inhibit cellular immunity ., partly explaining why ganglioside vaccines may not be an ideal IT approach. Furthermo
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Epidemiology of Primary Brain Tumorsexplosion of epidemiological and molecular genetic studies of BTs, has focused attention on this important human cancer, which, up until only a few decades ago, was relatively little studied. Despite this surge of interest, the etiology of the majority of nervous system tumors remains unknown. Inher
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Current Therapy for Primary Brain Tumorsto the eighth decade of life .,.. The average annual increase in the incidence of primary BTs in elderly patients in the United States, between 1974 and 1985, was 7.0% for the 75-79-yr-old age group, 20.4% in the 80–84 yr age group, and 23.4% in those 85 yr and older. These diagnoses were predominan
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Autologous Vaccine and Adoptive Cellular Immunotherapy as Treatment for Brain Tumorsi (such as anti-CD3) can activate primed T-lymphocytes to differentiate into Ag-specific effector cells in vitro, and interleukin-2 (IL-2) stimulates these cells to proliferate. Activated effector T-lymphocytes are able to travel to sites of tumor growth, enter and reject the tumor, and potentially
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Radiolabeled Antibodies for Therapy of Brain Tumorsxhibited Ab localization as detected by immunoscintigraphy .. Because of the nonspecificity of the target antigen (Ag), as well as the heterogeneous distribution of FIB in tumors, the utility of targeting FIB for diagnostic or therapeutic purposes was concluded to be low. In a series of studies ., D
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