Titlebook: Brain Hypothermia; Pathology, Pharmacol Nariyuki Hayashi (Professor) Conference proceedings 2000 Springer-Verlag Tokyo 2000 Trauma.brain.br

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Image Contrast and Pulse Sequences,ge because of its harmful complications and the lack of basic knowledge of its beneficial effects. In 1987, it was shown in animal experiments that lowering brain temperature only by a few degrees Celsius during ischemia could protect central neurons against ischemic damages. Since then, widespread

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https://doi.org/10.1007/978-1-4471-1413-0was complete destruction of neural tissue at the crush site, the area was filled in by other cells including neutrophils and connective tissue elements in both the hypothermia-treated and the untreated groups. These was a striking reduction in the progressive necrosis and cavitation that is characte

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Enhanced Neuronal Damage in Severely Brain-Injured Patients by Hypothalamus, Pituitary, and Adrenal s a certain time window. The initial stages of brain injury involve destruction of the brain tissue, localized brain ischemia, cytokine inflammation, and synaptic dysfunction with release of vascular agonists, catecholamines, dopamine, neurogenous agonists such as choline, excitatory amino acids, an

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Mild Hypothermia Amelioration of Damage During Rat Spinal Cord Injury: Inhibition of Pathological Miry. A marked proliferation of positive isolectin-staining microglia was observed in the control animals from 48 h after the injury. On the other hand, in both hypothermia-treated and sham animals, a small number of microglia were observed up to 72 h after the injury. These results suggest that mild