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Titlebook: Brain Edema XVI; Translate Basic Scie Richard L. Applegate,Gang Chen,John H. Zhang Book 2016 Springer International Publishing Switzerland

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Sevoflurane Preconditioning Confers Neuroprotection , Anti-apoptosis Effectsptotic Bcl-2 family proteins over pro-apoptotic proteins and with decreased activation of JNK and p53 pathways. Thus, our data suggest that suppression of apoptotic cell death contributes to the neuroprotection against ischemic brain injury conferred by sevoflurane preconditioning.
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Remote Ischemic Postconditioning (RIPC) After GMH in Rodentsproved cognition (MWM) and attenuated neuropathological ventricular enlargement (hydrocephalus) in juvenile animals following GMH. RIPC is a safe and noninvasive approach that improved sensorimotor and neuropathological outcomes following GMH in rats. Further studies are needed to evaluate for mecha
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Raúl Leal Ascencio,Marco Perez Cisnerosl thickness: mouse, rat, rabbit, and human..Our study demonstrates that NILT differentially penetrates the skulls. There is especially extensive attenuation of light energy penetration across the human calvaria, compared with animal skulls, which suggests that the power density setting used in strok
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https://doi.org/10.1007/3-540-46016-0the intervention. Continued efforts to complete the ongoing phase II study and definitive efficacy studies are needed to bring a candidate pharmacotherapy to a population of severe stroke patients that currently have no alternative.
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https://doi.org/10.1007/978-94-011-3676-1proved cognition (MWM) and attenuated neuropathological ventricular enlargement (hydrocephalus) in juvenile animals following GMH. RIPC is a safe and noninvasive approach that improved sensorimotor and neuropathological outcomes following GMH in rats. Further studies are needed to evaluate for mecha
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https://doi.org/10.1007/978-1-349-14529-4ical injury were impaired in the water maze, but performed better than animals with cortical injuries. However, only animals with cortical injuries were impaired on the rotarod. Additional covariates, such as edema and lesion size, may further clarify these phenotypes. Overall, we provide evidence t
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