Titlebook: Brain Edema; Proceedings of the S Yutaka Inaba,Igor Klatzo,Maria Spatz Conference proceedings 1985 Springer-Verlag Berlin Heidelberg 1985 H

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Martin Trauth,Elisabeth SillmannThe white matter constitutes the major site of fluid accumulation in brain edema. The myelinated axon is one of the major elements in the white matter. It is associated with five extracellular compartments, which normally are not easily recognizable, but are clearly demonstrable in various edematous conditions.

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Exudation of Plasma Protein Fractions in Vasogenic Brain EdemaThe passage of plasma proteins into the brain is pertinent to the question of resolution of vasogenic brain edema, as the proteins give rise to the colloidosmotic pressure of brain interstitial fluid, one of the parameters in the Starling relation of transcapillary fluid exchange [6].

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Dynamics of Fluid Movement Through Brain Parenchyma and into the CSF in Vasogenic Brain EdemaPrevious studies have shown that vasogenic edema spreads in the white matter by bulk flow and not by diffusion [1, 6, 8–12]. On the basis of the pressure-volume curve of the brain extracellular space in edema [2, 7, 9] it was concluded that fluid mobility must be significantly greater in the edematous than in the normal brain.

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Semiquantitative Evaluation of Interendothelial Cleft Changes in Water IntoxicationIt is generally held that the processes responsible for the selective permeability of the blood-brain barrier occur in the endothelium of brain microvasculature [1, 5, 11]. The precise mechanisms involved remain somewhat unclear; a variety of transendothelial transport mechanisms have been proposed [12, 13].

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Visualizing 3D Data in Earth Sciences,reasingly recognized. These are: (a) . with patterns of selective damage to various neuronal units following an ischemic insult; (b) . with regard to various parameters of ischemic injury; (c) the ., i.e., progressive development of the lesions following an ischemic insult, and related to it; and (d