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Titlebook: Bovine Spongiform Encephalopathy; The BSE Dilemma Clarence J. Gibbs Book 1996 Springer-Verlag New York, Inc. 1996 BSE.biochemistry.genetics

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https://doi.org/10.1007/978-3-540-89226-7n supplement (2). A subsequent case-control study of calf-feeding practices substantiated this hypothesis (3), and action to prevent further exposure from the food-borne source was taken in July 1988 when the feeding of ruminant derived protein to ruminants was banned.
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,Übergang: Therapieabschluss und Nachsorge, significant aspects of research efforts to define and evaluate the nature of vertical and horizontal transmission are presented. These include diagnosis and diagnostic problems, preclinical and subclinical infection, detection of scrapie agent in tissues of the reproductive tract, and the effect of different levels of exposure.
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Bovine Spongiform Encephalopathy Distribution and Update on Some Transmission and Decontamination St (2). Subsequently it was reported that clinical cases of BSE, unrecognized at the time, had probably occurred as early as April 1985 and that the origin of infection, in the first 190 cases or so, was feed in the form of ruminant-derived meat and bone meal (MBM) (3). Nothing has occurred since to alter this view (4).
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Recent Observations on the Epidemiology of Bovine Spongiform Encephalopathyn supplement (2). A subsequent case-control study of calf-feeding practices substantiated this hypothesis (3), and action to prevent further exposure from the food-borne source was taken in July 1988 when the feeding of ruminant derived protein to ruminants was banned.
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Scrapie: Studies on Vertical and Horizontal Transmission significant aspects of research efforts to define and evaluate the nature of vertical and horizontal transmission are presented. These include diagnosis and diagnostic problems, preclinical and subclinical infection, detection of scrapie agent in tissues of the reproductive tract, and the effect of different levels of exposure.
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Wie Objektempathie erlernt werden sollodendrocytes; (b) reactive gliosis; and (c) variable extracellular amyloid deposits, formed by the scrapie (CJD) isoform of prion protein, PrP., and/or prion protein (scrapie amyloid), PrP27-30 (1, 2).
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