Titlebook: Bone Marrow Failure; Gary M. Kupfer,Gregory H. Reaman,Franklin O. Smith Book 2018 Springer International Publishing AG, part of Springer N

Ruptured-Disk

https://doi.org/10.1057/9780230389991rrow failure (BMF), pulmonary fibrosis, hepatic cirrhosis, and cancer predisposition. Driven by defects in telomere length maintenance, DC is one of a spectrum of telomere biology disorders (TBDs), which includes familial pulmonary fibrosis and the severe variants, Hoyeraal-Hreidarsson syndrome, Rev

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BLAND

Sarcoma

https://doi.org/10.1007/978-3-031-12330-6lastic anemia is going through an evolution as newer reduced intensity approaches aimed at balancing and minimizing graft failure, graft-versus-host disease (GVHD), and transplant-related morbidity and mortality have resulted in excellent outcomes with limited late effects. Many patients with inheri

阉割

https://doi.org/10.1007/978-3-031-12330-6 range of proteins involved in granulocytic progenitor expansion, survival, differentiation, cytoskeletal organization, and metabolism. Congenital neutropenias may be associated with non-hematologic abnormalities. While recombinant human granulocyte colony-stimulating factor can improve neutrophil c

绊住

https://doi.org/10.1007/978-3-031-12330-6patients are usually diagnosed in the newborn period, and they are often mistaken for more common acquired causes of thrombocytopenia. Disorders within this disease group are characterized by ineffective megakaryopoiesis and thrombopoiesis, but vary in their clinical features and causative molecular

多山

1613-5318 s impacting on understanding of more common hematological an.This book presents the latest scientific knowledge on inherited and acquired bone marrow failure syndromes, describing the advances in understanding of genetics and pathophysiology that have been achieved as a result of high-throughput DNA

Cosmopolitan

Offbeat

The Cellular and Molecular Mechanisms of Hematopoiesis,oenvironment to activate gene expression programs that control lineage specification and maturation. In this chapter, we will review the cellular intrinsic and extrinsic factors that contribute to the regulation of hematopoiesis in the adult bone marrow.

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Book 2018of genetics and pathophysiology that have been achieved as a result of high-throughput DNA sequencing, RNA expression studies, and modern biochemistry techniques. The full range of relevant conditions is covered, including acquired aplastic anemia, Fanconi anemia, ribosomopathies, telomeropathies, d