Titlebook: Blood-Brain Barrier Permeability Changes after Subarachnoid Haemorrhage: An Update; Clinical Implication Antonino F. Germanò,Francesco Toma

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Evaluation of Allograft Fixationid haemorrhage. Nimodipine is a highly lipid-soluble dihydropyridine derivative that readily crosses the blood-brain barrier. In animal studies, nimodipine has been shown to be effective in increasing cerebral blood flow; preventing vasoconstriction attributable to sympathetic stimulation, hypocapni

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Overview: First monograph on blood-brain barrier changes after cerebral bleeding.Balanced consideration of basic science and clinical literature.Provides strategies for pharmacological interventions978-3-7091-7250-6978-3-7091-6194-4

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Greig Cephalopolysyndactyly Syndromeion, massive increase in intracranial pressure with a consequent decrease in cerebral perfusion pressure and tissue ischaemia, local increase in tissue pressure with microvascular compression, or the effects of vasoactive factors within the cerebrospinal fluid released from blood breakdown products.

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Timothy E. Yates,Sreekumar Madasseryonsequences could only be inferred from the displacements of vessels or ventricles they provoked, and only exceptionally did they manifest themselves. But since the introduction of radio-isotope scanning brain lesions have revealed their presence on account of barrier breakdown causing extravasation of the radioactive tracer.

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Antonino F. Germanò,Francesco TomaselloFirst monograph on blood-brain barrier changes after cerebral bleeding.Balanced consideration of basic science and clinical literature.Provides strategies for pharmacological interventions

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BBB Dysfunction after SAH,ion, massive increase in intracranial pressure with a consequent decrease in cerebral perfusion pressure and tissue ischaemia, local increase in tissue pressure with microvascular compression, or the effects of vasoactive factors within the cerebrospinal fluid released from blood breakdown products.

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