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Titlebook: Blood Pressure and Arterial Wall Mechanics in Cardiovascular Diseases; Michel E. Safar,Michael F. O‘Rourke,Edward D. Froh Book 2014 Spring

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https://doi.org/10.1007/978-94-007-6220-6(DC) and cross-sectional compliance (CC) can be calculated. Distensibility is a measure of wall elasticity and the inverse of stiffness. Compliance reflects the buffering capacity of the artery. Local stiffness assessment is the only method that can assess both wall properties. In addition, if the p
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https://doi.org/10.1007/978-3-658-29723-7unctional elements such as the scleroprotein elastin, an essential component of elastic fibres. This deterioration is the basis of many important pathological conditions in man such as stiffening of the arterial wall..In this article we will examine how ageing can modify elastin and the role of calc
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https://doi.org/10.1007/978-3-658-29723-7ffening. Available data on genome-wide association (GWA) have been initiated on PWV and have identified common genetic variation in specific loci or single-nucleotide polymorphisms (SNP) significantly associated with PWV. Telomere length at birth is strongly determined genetically and is the main de
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https://doi.org/10.1007/3-540-37672-0duction in distensibility of muscular arteries shown under particular conditions of stress, such as exposure to high altitude and, in general, to hypoxia. Changes in sympathetic activity may be influenced by baroreflex regulation of cardiovascular homeostasis. Reflex changes of arterial tone and mod
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Large Arteries, Microcirculation, and Mechanisms of Hypertensionl function of the cardiovascular system. Extrinsic neurohormonal control systems contribute to this match. In addition, blood pressure transfers energy, mechanical signals, and metabolic factors independently of these neurohormonal control mechanisms. The plasticity of vascular structure provides a
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Ventricular-Arterial Coupling and Mechanism of Wave Reflectionsventricular and arterial elastance (analysis in pressure-volume plane) and (ii) a novel approach based on assessment of time-varying myocardial stress. The latter analysis, in the time domain, allows to directly link left ventricular myocardial stress with systemic arterial properties and with the m
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