Titlebook: Blistering Diseases; Clinical Features, P Dédée F. Murrell Book 2015 Springer-Verlag Berlin Heidelberg 2015 Epidermolysis bullosa.Immunoflu

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描述

Design und Auswertung einer Anwendungsstudieclude specialized laminins, collagens, integrins, as well as other unique structural components which together serve to connect the intermediate filament network of the basal keratinocyte cytoskeleton to the interstitial collagen network of the papillary dermis. Damage to the dermal-epidermal baseme

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pacific

Design und Auswertung einer Anwendungsstudieave been shown to regulate additional biological processes of importance such as protein synthesis, cell migration, and apoptosis. Much of this knowledge has been gained through the study of rare disorders, known as keratinopathies, which are caused by mutations in genes encoding the various epiderm

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Öffentlicher Raum als privater Bildungsraumosome anchoring complexes. Over the last few years, however, the clinicopathological spectrum of EB has been expanded to also include primary abnormalities in desmosome junctions. Desmosomes are intercellular junctions that contribute to cell-cell adhesion, signalling, development and differentiatio

intrude

Wolfgang Wittwer,Andreas Diettrichere are several variants of pemphigus, each with unique clinical, histological, and immunologic features. Interestingly, when different desmosomal proteins are targeted by the autoimmune response, different clinical and histological features are seen. In this chapter we review the key intercellular

Toxoid-Vaccines

Wolfgang Wittwer,Andreas Diettrichthelial cells to the underlying basement membrane. It is a homotrimeric type II transmembrane protein consisting of three 180 kDa alpha-1 (XVII) chains. Each individual chain is encoded by the . gene. Mutations in . usually lead to loss of collagen XVII and a phenotype comprising congenital generali