Titlebook: Bipolar Depression: Molecular Neurobiology, Clinical Diagnosis, and Pharmacotherapy; Carlos A. Zarate Jr.,Husseini K. Manji Book 2016Lates

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Neuroimaging Studies of Bipolar Depression: Therapeutic Implicationsargement of the third and lateral ventricles are likely associated with cerebrovascular disease. Mood stabilizers and antidepressant drugs may attenuate pathological limbic activity, increase neurotrophic processes, and decrease inflammation, restoring balance to the system.

REP

Pharmacological Treatment of Acute Bipolar Depressionlanzapine plus fluoxetine (OFC), modafinil, armodafinil, lithium, valproate, carbamazepine, ketamine, .-acetyl cysteine (NAC), inositol, pramipexole, riluzole, and standard antidepressants, among others. Currently, five medications are supported by Category A criteria. In order of the strength of su

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Pharmacological Treatment of the Maintenance Phase of Bipolar Depression: Efficacy and Side Effectssults and cumulative risk for relapse/recurrence showed that only quetiapine demonstrated bimodal efficacy in preventing both depressive and manic/mixed/hypomanic relapses. The other medications were only efficacious for preventing manic/mixed relapses or more effective in preventing manic/mixed rel

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tympanometry

Decolonisation and Legitimation in Nigeriancorporation of BD into the modern operationalised diagnostic criteria such as the Research Diagnostic Criteria and DSM-III in the late twentieth century. In considering this history of the concept of BD, several broad themes become apparent. First, the consistency of the clinical descriptions stand

留恋

https://doi.org/10.1007/978-1-349-12342-1e is therefore best addressed in relation to the individual’s prototypic episodes. The high rates of nonpsychotic and psychotic melancholic depression in those with BD invite consideration as to whether such episodes differ from similar states experienced by those with equivalent unipolar states. Se

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TOXIN

Legitimacy in International Lawargement of the third and lateral ventricles are likely associated with cerebrovascular disease. Mood stabilizers and antidepressant drugs may attenuate pathological limbic activity, increase neurotrophic processes, and decrease inflammation, restoring balance to the system.