Titlebook: Biomarkers in Kidney Disease; Vinood B. Patel,Victor R. Preedy Reference work 2016 Springer Science+Business Media Dordrecht 2016 Applicat

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https://doi.org/10.1007/978-3-322-84086-8nd can induce the onset of a specific disease and on the other hand may represent potential biomarkers for the diagnosis and therapy. In this chapter, the miRNA functions and modulations are described in several kidney diseases such as polycystic kidney disease, primary and secondary glomerulonephri

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,Gerechtigkeit im Generationenverhältnis,on-carrying molecules which are essential to bacterial and eukaryotic cells to meet their needs of this mineral and then ensure survival. Indeed, NGAL primarily acts as an innate nonspecific antibacterial factor, since it prevents bacteria to internalize iron thus inhibiting their growth. However, s

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https://doi.org/10.1007/978-3-642-72517-3tative biomarkers, chemokines emerge as promising molecules since they play relevant roles in the pathophysiology of chronic kidney disease. In pediatric patients, glomerular diseases and congenital anomalies of the kidney and urinary tract are the commonest cause of chronic kidney disease. The eval

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Klaus Jürgen Husfeldt,Franz-Josef Rothnd easily measured either by blood or urine. Unfortunately, such an ideal marker, as yet, does not exist. Different pathological states may increase the level of a tumor marker in the absence of any neoplasia, or alternatively during these conditions, not every subject with cancer has abnormally hig

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Die Subclavia-Carotis-Transposition) is likely the most important factor in the development and maintenance of hypertension..Recently, two distinct RASs were identified: first, the systemic or circulatory RAS and, second, the local or tissue-specific RAS. The local RAS is found in many tissues such as those of the heart, brain, kidne

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https://doi.org/10.1007/978-3-642-72517-3r for cardiovascular disease (CVD). Its silent development, together with the lack of specific and early accessible indicators of renal damage, often results in a late diagnosis when kidney damage is irreversible. Omics approaches (genomics, proteomics, metabolomics) account with the advantage of in