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Titlebook: Biology of Renal Cell Carcinoma; Ronald M. Bukowski,James H. Finke,Eric A. Klein Conference proceedings 1995Latest edition Springer-Verlag

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期刊全称Biology of Renal Cell Carcinoma
影响因子2023Ronald M. Bukowski,James H. Finke,Eric A. Klein
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图书封面Titlebook: Biology of Renal Cell Carcinoma;  Ronald M. Bukowski,James H. Finke,Eric A. Klein Conference proceedings 1995Latest edition Springer-Verlag
影响因子.Biology of Renal Cell Carcinoma. presents the proceedings of the Third International Symposium on the Biology of Renal Cell Carcinoma, March 1994. This symposium gathers "internationally acclaimed researchers from relevant fields of renal cell carcinoma to present their latest work and to interact on the interpretation of these findings and direction for subsequent studies. The program is directed to physicians and research scientists involved in the clinical and basic research aspects of the treatment of renal cell carcinoma." (from the Symposium Announcement).Topics covered in the areas of Molecular Biology of Renal Cell Carcinoma: Cytogenetics, Genetic Approaches to Therapy of Renal Cell Carcinoma, Immunology of Renal Cell Carcinoma - T-Cell Anergy, New Approaches with Renal Cell Carcinoma - Cytokines.
Pindex Conference proceedings 1995Latest edition
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Alterations in Signal Transduction in T Cells from Cancer Patientsi-tumor effect. These approaches have been incorporated in human immunotherapy trials. Low dose cyclophosphamide has been administered before the transfer of activated cells in an attempt to block “suppressor cells” and hopefully improve the anti-tumor response. Similarly, sublethal irradiation appe
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Impaired Signal Transduction in Tumor Infiltrating T Cells from Patients with Renal Cell Carcinomag events that include the formation of second messengers and the activation of serine/threonine kinases along with phosphatases which are important for downstream gene expression (4). IL2 binding to its receptor is an important step in T cell activation and proliferation (10). This receptor is a tri
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T-Lymphocyte Response in Renal Cell CarcinomaCC TIL nor PBMC cultured with IL-2 alone displayed tumor-specific cytotoxicity. (1–8) These results suggest that RCC TIL have little or no CTL precursors, possibly because of the lower immunogeneity of untreated RCC cells. Poorly immunogenic animal tumor cells can be converted into highly immunogeni
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Kompendium der Mediengestaltungor genes in most human cancers (3). However, since tumor suppressor genes, like oncogenes, encode the proteins essential for cell differentiation and proliferation, both genes may be involved in the same control mechanism which regulates normal cell growth.
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Kompendium der Mediengestaltungg events that include the formation of second messengers and the activation of serine/threonine kinases along with phosphatases which are important for downstream gene expression (4). IL2 binding to its receptor is an important step in T cell activation and proliferation (10). This receptor is a tri
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