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Titlebook: Biology of Chlamydia; Georg Häcker Book 2018 Springer International Publishing AG, part of Springer Nature 2018 Chlamydia developmental cy

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Book 2018cterial agent of sexually transmitted disease. This volume reviews current knowledge of chlamydial biology, covering the unusual structure of the bacteria – which alternate between metabolically almost inactive and fast-dividing forms. It also discusses the ways in which .Chlamydia. manipulates the
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https://doi.org/10.1007/978-3-658-22665-7s studied so far is the host cell cytoskeleton, with past and recent findings revealing crucial roles in invasion, inclusion maintenance, nutrient acquisition, and egress. The molecular details of how . co-opts the cytoskeleton is becoming clearer, with bacterial factors and their corresponding host cell targets identified.
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Deconstructing the Chlamydial Cell Wall,re responsible for many acute and chronic diseases in humans, and .-related bacteria are emerging pathogens. We revisit past efforts to detect cell wall material in . and .-related bacteria in the context of recent breakthroughs in elucidating the underlying cellular and molecular mechanisms of the
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Manipulation of the Host Cell Cytoskeleton by ,, elementary body and the vegetative, but non-infectious reticulate body. . spends a significant portion of its development in the non-infectious stage, demanding an effective strategy of manipulating the host cells to ensure its intracellular survival and replication. A common target of all . specie
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Subversion of Cell-Autonomous Host Defense by , Infection,nside a eukaryotic cell and the lack of any extracellular niche, they have to perfectly adapt to compartmentalized intracellular environment of the host cell and counteract the numerous defense strategies intrinsically present in all eukaryotic cells. This so-called cell-autonomous defense is presen
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The Hidden Genomics of ,iches, and at the -species, -serotype or even -genus level, and in local, national and global settings. This has been instrumental in cataloguing and revealing a huge a range of diversity in this bacterium, when at first we thought there was little. Genomics has challenged assumptions, added insight
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as the Cause of Infectious Infertility: Acute, Repetitive or Persistent Long-Term Infection?,citis and if ascending to the upper FGT may result in serious sequelae such as pelvic inflammatory disease (PID), salpingitis and tubal factor infertility (TFI). The factors leading to this complication are still not completely understood. We elaborate four different models for host–pathogen interac
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