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Titlebook: Biology of Brain Dysfunction; Volume 1 Gerald E. Gaull Book 1973 Plenum Press, New York 1973 biology.brain.growth.information.molecular bio

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期刊全称Biology of Brain Dysfunction
期刊简称Volume 1
影响因子2023Gerald E. Gaull
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图书封面Titlebook: Biology of Brain Dysfunction; Volume 1 Gerald E. Gaull Book 1973 Plenum Press, New York 1973 biology.brain.growth.information.molecular bio
影响因子The growth of neurochemistry, molecular biology, and biochemical genetics has led to a burgeoning of new information relevant to the pathogenesis of brain dysfunction. This explosion of exciting new information is crying out for collation and meaningful synthesis. In its totality, it defies systematic summa­ tion, and, of course, no one author can cope. Thus invitations for contributions were given to various experts in areas which are under active investigation, of current neurological interest, and pregnant. Although this project is relatively comprehensive, by dint of size, other topics might have been included; the selection was solely my responsibility. I believe systematic summation a virtual impossibility-indeed, hardly worth the effort. The attempt to assemble all of the sections involved in a large treatise with multiple authors inevitably results in untoward delays due to the difference in the rate at which various authors work. Therefore, the following strategy has been adopted: multiple small volumes and a relatively flexible format, with publication in order of receipt and as soon as enough chapters are assembled to make publication practical and economical. In this wa
Pindex Book 1973
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Kommunikation und Kommunikationsmodelle,mia” means simply “galactose in the blood.” In animals, galactosemia can be achieved by overloading the organism with this sugar by feeding a diet rich in galactose. In man, one has encountered two types of congenital disease, both called “galactosemia.” One type of hereditary galactosemia is caused
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https://doi.org/10.1007/3-8350-5717-0studies in humans, and serial determinations are difficult in animals. On the other hand, CSF can be sampled serially, ., without altering physical or chemical parameters. Therefore, the CSF’s dynamic alterations are used to define dysfunction within the CSF compartment, as a direct index of dysfunc
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Kommunikation von Unternehmertum biting behavior. These patients have hyperuricemia and thus may have any of the clinical manifestations of gout, including urinary tract stone disease, nephropathy, arthritis, and tophi. Activity of the enzyme hypoxanthine-guanine phosphoribosyl transferase is virtually absent.
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