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Titlebook: Biological and Hormonal Therapies of Cancer; Kenneth A. Foon,Hyman B. Muss Book 1998 Springer Science+Business Media New York 1998 Tumor.a

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Hormonal strategies for the prevention of breast cancer,opausal women. Over the intervening years, various hormonal manipulations have been developed to treat earlier stages of breast carcinoma. Favorable results from these interventions, in combination with a growing understanding of the biology of breast carcinoma, have fostered the hope that hormonal manipulations may prevent breast cancers.
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https://doi.org/10.1007/978-3-662-61103-6ogy for analysis, and the apparently minimal effects seen in these trials led to a loss of interest in this modality, which was exacerbated by the promising early results of adjuvant chemotherapy in the mid 1970s [.-.].
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Cellular vaccine therapies for cancer,n grow progressively in the presence of concommitant immunity) that until recently seems to have gone unnoticed. Investigators have narrowly focused on improving the immunogenicity of their vaccines and are only now coming to recognize the emerging evidence that human tumors can and do grow in the presence of an immune response.
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Ovarian ablation as adjuvant therapy for early-stage breast cancer,ogy for analysis, and the apparently minimal effects seen in these trials led to a loss of interest in this modality, which was exacerbated by the promising early results of adjuvant chemotherapy in the mid 1970s [.-.].
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The regulation of estrogen receptor expression and function in human breast cancer,le in normal mammary development was demonstrated using transgenic mice that were homozygously deleted for the gene. Adult female ER knockout mice develop mammary glands with only vestigial ducts present at the nipples [.].
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Book 1998 concerning biologic and hormonal therapy of cancer. We have attempted to provide the reader with topics of major interest in a timely fashion. There is renewed interest in biologic therapy of cancer. Two chapters review the role of interferon in the hematologic malignancies and in solid tumors. Vac
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Interferon therapy of hematologic malignancies,s such as glucose 6-phosphate dehydrogenase (G6PD) in heterozygous individuals with CML, X-chromosome DNA restriction fragment-length polymorphism of hypoxanthine glucuronyl ribosyl phosphoryl transferase, and the presence of the Philadelphia (Ph’) chromosome and other clonal cytogenetic markers [.-.].
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