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Titlebook: Biological Effects of Alcohol; Henri Begleiter Book 1980 The Editor(s) (if applicable) and The Author(s), under exclusive license to Sprin

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Formation of Isoquinoline Alkaloids: Effect of Time and Route of Administration of Ethanolaining substances to form Schiff’s bases that then undergo cyclisation (Kapadia and Fales, 1968; Leete and Braunstein, 1969), but these were the first indications that acetaldehyde could alter mammalian neuroamine metabolism in an analogous, biosynthetic fashion.
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Altered Response to Cessation of Nerve Impulse Flow in Dopamine Neurons After Chronic Ethanol Adminil develop at or beyond GABA receptors following chronic ethanol treatment. Preliminary findings concerning the effects of chronic ethanol administration on dopamine autoreceptor sensitivity are also reported.
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Stand der Wissenschaft und Technik,of diastereomeric 4,6,7-trihydroxy TIQ or their optical isomers. The preparation of TIQ alcohols 1. and 2. (Figure 1) belonging to this class of compounds, and prepared from (±)-NE and formaldehyde and (-)-E and acetaldehyde respectively, has been reported.
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Calcium Receptor Binding in Synaptic Membranes of ICR, C57, DBA Mice After Ethanol Exposurey of membrane components (1,2,3). Implicit in these assumptions is the fact that ethanol must act to alter structural components in the membrane which directly or indirectly influence membrane function. Indeed both lipid and protein components have been shown to be altered after ethanol exposure (4,5,6).
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