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Titlebook: Biochemistry of Inflammation; J. T. Whicher,S. W. Evans Book 1992 Springer Science+Business Media Dordrecht 1992 biochemistry.cytokine.inf

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发表于 2025-3-21 18:35:02 | 显示全部楼层 |阅读模式
期刊全称Biochemistry of Inflammation
影响因子2023J. T. Whicher,S. W. Evans
视频video
学科分类Immunology and Medicine
图书封面Titlebook: Biochemistry of Inflammation;  J. T. Whicher,S. W. Evans Book 1992 Springer Science+Business Media Dordrecht 1992 biochemistry.cytokine.inf
影响因子Our understanding of inflammation has increased rapidly inrecent years, due in large part to the impact of molecular biology andgene identification and cloning. This book brings together ideas froma number of different biochemical disciplines which are frequently notintegrated. .The first chapter gives a visual overview of the subject; theremaining chapters are organized into three themes: the affectormolecules, the regulatory components and the processes of inflammationitself. .This book is essential reading for the busy physician or pathologistwho wants to be up-to-date with the latest developments in immunologyas they affect the diagnosis and treatment of many conditions. .
Pindex Book 1992
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发表于 2025-3-21 21:08:26 | 显示全部楼层
The kinin system: current concepts and perspectives,on of kinins is increased in the initial phase of the inflammatory process both in experimental animals. and in humans.. Despite their rapid inactivation by a variety of peptidases, a local generation of kinins may be maintained during the subacute and chronic phase of inflammation by the continuous
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Role of cellular proteinases and their protein inhibitors in inflammation, rubor, calor, and turgor are all dependent for their production on the action of proteolytic enzymes. There is no possibility of reviewing all of the proteinases involved in inflammation in a single chapter. There are literally scores of enzymes that would have to be considered. In order to reduce
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Eicosanoids (prostaglandins and leukotrienes),gues that aspirin and other non-steroidal anti-inflammatory drugs (NSAIDs) inhibit PG synthesis.. More recently, it has been suggested that other products of arachidonic acid metabolism, such as leukotrienes, are also mediators of allergic and inflammatory reactions. Inhibitors of leukotriene synthe
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Glucocorticoids and lipocortin,s this is predominantly hydrocortisone (cortisol), but other animals may release different but related steroids. For a long while it was believed that steroids were in some way involved in the mounting of the response to stress (such as an inflammatory response), but it has only recently become clea
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Platelet activating factor and cytokine feedback regulation of endothelial integrity; implications , while immune functions prevent invasion by foreign organisms. These two processes are closely linked as many of the same cells and molecules are operative in both. In a normal controlled response following injury, polymorphonuclear leukocytes (PMNL) leave the flowing blood and adhere to the endoth
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Tumour necrosis factor (TNF) and inflammation,, but nevertheless essential, aspect of these defences is the inflammatory response. In contrast to the immune responses involving antibodies and effector T lymphocytes, inflammation is relatively non-specific, but it does provide a quick and usually effective anti-invasive response. The inflammator
发表于 2025-3-23 09:32:33 | 显示全部楼层
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