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Titlebook: Biochemistry of Copper; Maria C. Linder Book 1991 Springer Science+Business Media New York 1991 Absorption.biochemistry.chemistry.metaboli

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Book 1991nterpret where we currently stand in our knowledge of the structure, function, regulation, and metabolism of Cu-dependent processes and sub­ stances, especially proteins. Thus, I have tried to make this a source book for historic as well as current information on all aspects of copper bio­ chemistry
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been to interpret where we currently stand in our knowledge of the structure, function, regulation, and metabolism of Cu-dependent processes and sub­ stances, especially proteins. Thus, I have tried to make this a source book for historic as well as current information on all aspects of copper bio­ chemistry978-1-4757-9434-2978-1-4757-9432-8
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https://doi.org/10.1007/978-3-8349-9527-8an Barnefeld and Van den Hamer (1984), on the other hand, found 22% of an oral dose in the (washed) stomach wall of mice after 2 h, of which none remained at 6 h. The earlier data of Van Campen and Mitchell (1965) for rats also suggested that there is a lower rate of absorption of copper, the farthe
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,Kostenträgerrechnung mit Teilkosten,that histidine (4–12 .) stimulated uptake of .Cu (10 .) by rat liver or kidney slices (or by Ehrlich ascites tumor cells) when incubated in a Krebs—Ringer medium containing 4% albumin. Harris and Sass-Kortsak (1967) showed that there was the same degree of uptake (and stimulation of uptake) by amino
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,Kostenträgerrechnung mit Teilkosten,rcentages of total tissue copper in the cytosolic fractions, about 65% in the rat and about 80% in the human. Pig liver may have a lower percentage in the cytosol (about 40%) and a larger percentage (35%) in the nuclear fraction (Owen .., 1977).
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Absorption of Copper from the Digestive Tract,an Barnefeld and Van den Hamer (1984), on the other hand, found 22% of an oral dose in the (washed) stomach wall of mice after 2 h, of which none remained at 6 h. The earlier data of Van Campen and Mitchell (1965) for rats also suggested that there is a lower rate of absorption of copper, the farthe
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Copper Uptake by Nongastrointestinal Vertebrate Cells,that histidine (4–12 .) stimulated uptake of .Cu (10 .) by rat liver or kidney slices (or by Ehrlich ascites tumor cells) when incubated in a Krebs—Ringer medium containing 4% albumin. Harris and Sass-Kortsak (1967) showed that there was the same degree of uptake (and stimulation of uptake) by amino
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Extracellular Copper Substituents and Mammalian Copper Transport, plasma but also in interstitial fluid and in other fluids involved with copper transport, such as the cerebrospinal fluid in the brain (on the other side of the blood—brain barrier) and fluids in specialized areas (testes, amniotic fluid). This chapter reviews what is known about these components,
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