Titlebook: Biochemical Regulation of Myocardium; Roland Vetter,Ernst-Georg Krause Book 1996 Kluwer Academic Publishers 1996 ATP.Calcium.Glycogen.Lipi

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phosphorylation of the cardiac L-type calcium channel beta-subunit in response to catecholaminesett 335: 217–222, 1993). We have assessed the identity of the β-subunit as well as its . phosphorylation in representative experimental groups of catecholamine-challenged canine hearts. Adrenergic stimulation by high doses of both noradrenaline and isoprenaline induced rapid (within 20 sec) and near

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Early after-depolarisations induced by noradrenaline may be initiated by calcium released from sarcod with Indo 1-AM. Membrane potential was recorded by means of the patch electrode and patch amplifier set to the currentclamp mode. Cells were stimulated at a rate of 30/min by 3 ms pulses of the current injected through the recording electrode. Superfusion of NA resulted in slight shortening of act

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Cardiac pump function of the isolated rat heart at two modes of energy deprivation and effect of adr by disturbances either in mitochondrial ATP production or ATP-phosphocreatine transformation. Amytal (0.3 mM), an inhibitor of mitochondrial respiration, or iodoacetamide (., 0.1 mM) reducing in this dose creatine kinase activity to 19% of the initial level, were used, respectively. Myocardial ATP

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Regulation of β-adrenoceptor properties and the lipid milieu in heart muscle membranes during stressrenoceptors during various forms of stress or chronic adrenergic stimulation. Analysis of the fatty acid profile of phosphatidylcholine (PC) and phosphatidylethanolamine (PE) in sarcolemma or cardiac muscle membranes showed partial replacement of 18:2n-6 by 20:4n-6 in PC and replacement of both 18:2

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