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Titlebook: Benign Hematologic Disorders in Children; A Clinical Guide Deepak M. Kamat,Melissa Frei-Jones Book 2021 Springer Nature Switzerland AG 2021

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期刊全称Benign Hematologic Disorders in Children
期刊简称A Clinical Guide
影响因子2023Deepak M. Kamat,Melissa Frei-Jones
视频video
发行地址Presented in a clear and concise format for ease of use.Features over 100 illustrations, photographs, and tables.Written by experts in the field
图书封面Titlebook: Benign Hematologic Disorders in Children; A Clinical Guide Deepak M. Kamat,Melissa Frei-Jones Book 2021 Springer Nature Switzerland AG 2021
影响因子This book provides a comprehensive overview of benign hematologic disorders in children. Divided into nine sections, the text reviews common hematologic disorders or conditions that affect children, while providing state-of-the-art information on pathophysiology, diagnosis, treatment, and management strategies..The text begins with a section on hematopoiesis, and the next section covers red blood cell disorders. The following sections provide overviews of platelet disorders, white blood cell disorders, and coagulation disorders. The sixth and seventh sections discuss neonatal hematology and bone marrow failure syndrome. The eighth section reviews supportive care, while the final section covers miscellaneous subjects including pediatric vascular anomalies and complement dysregulation syndromes..Written by experts in the field, .Benign Hematologic Disorders in Children: A Clinical Guide .is a valuable resourcefor clinicians and practitioners who treat children afflicted with these disorders..
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Lead Poisoningtrated that there is no safe level of lead exposure in children. Despite efforts to reduce children’s exposure to lead, a substantial number of children continue to be exposed to lead through lead-contaminated dust, water, and soil. Lead affects all systems of the body, but it also affects the neuro
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Immune and Nonimmune Hemolytic Anemiaout 120 days in the circulation (Shemin and Rittenberg, J Biol Chem. 166:627–636, 1946). Hemolysis is therefore defined as reduced survival of circulating RBCs due to premature destruction. Under steady state conditions, the rate of RBC production equals the rate of RBC loss. As a compensatory mecha
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Thalassemials. Defects can arise in both the alpha and beta subunits with varying severity of symptoms. Currently most patients with severe phenotypes rely on lifelong chronic transfusions, leading to iron overload and risk for alloimmunization. New therapies are being investigated to decrease transfusion requ
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Disorders of RBC Metabolismphosphate shunt and glycolytic pathway producing adenosine triphosphate (ATP) and nicotinamide adenine dinucleotide (NAD), respectively. Reduction in enzyme activity in these pathways results in hemolysis. The two most common disorders of RBC metabolism are glucose-6-phosphate dehydrogenase (G6PD) d
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Immune Thrombocytopenialdhood ITP generally follows a viral illness. Diagnosis is made by a thorough history, physical examination, complete blood count (CBC) evaluation, and peripheral smear review to rule out other diagnoses. Additional laboratory testing is usually not necessary but may be done if the clinical scenario
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Inherited and Congenital Thrombocytopeniath bone marrow failure syndromes or other genetic disorders. Prompt recognition and diagnosis through algorithmic testing is key in order to provide anticipatory guidance, prevention of bleeding complications, and screening for associated syndromic medical comorbidities. Treatment is generally suppo
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