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Titlebook: Behavioral Neurobiology of PTSD; Eric Vermetten,Dewleen G. Baker,Victoria B. Risbro Book 2018 Springer International Publishing AG, part o

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期刊全称Behavioral Neurobiology of PTSD
影响因子2023Eric Vermetten,Dewleen G. Baker,Victoria B. Risbro
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发行地址Summarizes the latest studies in the neurobiology of long term consequences of trauma.Maximizes reader insights into the mechanisms of risk and resilience to stress.Treats both theoretical and applied
学科分类Current Topics in Behavioral Neurosciences
图书封面Titlebook: Behavioral Neurobiology of PTSD;  Eric Vermetten,Dewleen G. Baker,Victoria B. Risbro Book 2018 Springer International Publishing AG, part o
影响因子This volume focuses on the behavioral neuroscience that supports our understanding of the neurobiology of trauma risk and response.  The collection of articles focuses on both preclinical and clinical reviews of (1) state-of-the-art knowledge of mechanisms of posttraumatic stress disorder (PTSD) and co-occurring disorders, (2) the biological and psychological constructs that support risk and resiliency for trauma disorders, and (3), novel treatment strategies and therapeutics on the horizon.
Pindex Book 2018
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Integrating NIMH Research Domain Criteria (RDoC) into PTSD Research,nd debilitating disease. However, despite all research efforts, so far no drug that has specifically targeted PTSD core symptoms progressed to clinical use. Instead, although not overly efficient, serotonin re-uptake inhibitors continue to be considered the gold standard of PTSD pharmacotherapy. The
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Neurobiological Programming of Early Life Stress: Functional Development of Amygdala-Prefrontal Cirinvestigated the effects of early adversity on neurobiology in adulthood, developmental approaches can provide further insight on the neurobiological mechanisms that link early experiences and long-term mental health outcomes. In the current review, we discuss the role of emotion regulation circuitr
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Subanesthetic Dose Ketamine in Posttraumatic Stress Disorder: A Role for Reconsolidation During Trand its required molecular machinery can provide an opportunity to target pathological memories for emotionally arousing events. As memories become labile upon retrieval, novel information can update the strength and course of these consolidated memories. Targeting the process of reconsolidation may
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