Titlebook: Basic and Clinical Toxicology of Organophosphorus Compounds; Mahdi Balali-Mood,Mohammad Abdollahi Book 2014 Springer-Verlag London 2014 ch

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Toxicity and Novel Biomarkers of OP Exposure,erned with OP toxicity in humans. OPs cause four important neurotoxic effects, including the cholinergic syndrome, the intermediate syndrome, OP-induced delayed polyneuropathy, and chronic OP-induced neuropsychiatric disorder. Compared with the cholinergic syndrome, that causes millions of cases of

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languid

,Clinical Management of Organophosphorus Nerve Agents’ Poisonings,ily. They are highly lipophylic. NAs irreversibly inactivate acetylcholine esterase (AChE) and lead to accumulate toxic levels of acetylcholine. Respiratory system is the main organ that is affected by NAs and respiration may be arrested due to central and peripheral dysfunctions. NA exposure is det

ALIBI

Occupational and Environmental Aspects of Organophosphorus Compounds,and placing pesticides on the market are under national and international legislative framework, although in many developing countries the regulation is still inadequate or not properly enforced. Occupational exposure to OPs includes any job which involves either direct or indirect contact with OP c

Archipelago

Summary, Discussion, and Conclusions, of toxicity. A discussion of all basic and clinical aspects of OP compounds from their synthesis and toxicity, to novel biomarkers, epidemiology of poisoning, occupational and environmental exposure, risk assessment, clinical management and guidance on prevention and control have been covered in th

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exclusice

History of the Use and Epidemiology of Organophosphorus Poisoning,ion and chemical terrorism. The use of sarin in chemical terroristic attacks has been reported during 1994–1995 in Matsumoto and Tokyo subway lines, Japan..Today, a wide range of OPs with a variety of biological properties are available for agricultural and public health usages including insecticide

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镀金

Toxicity and Novel Biomarkers of OP Exposure,OP is tightly bound to egasyn, and subsequently, BG is dissociated and released into blood. Consequently, the increase of plasma BG activity is a good biomarker of OP exposure. The data presented in this chapter indicate the increase in plasma BG activity which is a much more sensitive biomarker to