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Titlebook: Basic Mechanisms of Physiologic and Aberrant Lymphoproliferation in the Skin; W. Clark Lambert,Benvenuto Giannotti,Willem A. Vlo Book 1994

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Eric Wiebelhaus-Brahm,S. Ra’phael Davishich may represent examples of such a progression are presented. Arguments for and against the hypothesis that large plaque parapsoriasis, retiform parapsoriasis, or both, should be classified as mycosis fungoides are discussed. It is the author’s opinion that large plaque parapsoriasis should not b
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https://doi.org/10.1007/978-3-031-47347-0n techniques such as immunohistochemistry and molecular biology (Wood et al., 1986) has shown that cutaneous T-cell lymphoma is a heterogenous group, with many clinicopathological subsets (Slater, 1991). The diagnostic criteria for certain specific forms of cutaneous T-cell lymphoma are reviewed wit
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Heritage in the Context of Globalizations represent a vexing problem when dealing with the initial phases of a lymphomatous process. Even advanced diagnostic techniques, like immunophenotyping, quantitative DNA cytophotometry, and molecular genetic analysis, have proven to be unsuitable for solving the problem; thus, light microscopy rema
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γδ T Lymphocytes in Mice and Man: A Reviewmake to the immune system?” We review here how TCR diversity is generated at the molecular level and what this means for the capability of TCR αβ and TCR γδ to recognize a variety of antigenic peptides in the context of conventional and alternative presenting molecules. We will discuss how tissue di
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Ontogeny, Features and Functions of Epidermal T Lymphocytesng to the T cell system (Stingi et al., 1989). In man, the majority of cutaneous T lymphocytes are found in the dermis, where they are preferentially clustered around postcapillary venules and around the appendages (Bos et al., 1987). Intraepidermal T cells account for only ≤ 10% of all T cells with
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Activation of Reactive Versus Malignant T Cells in Cutaneous T Cell Lymphoma: Role of Abnormal Antigin the biology of CTCL. T cell receptor gene rearrangement (TCRGR) analysis allows the identification of the malignant clone on the basis of the unique size of the TCR gene fragments after enzyme digestion. We identified the malignant clone by TCRGR analysis of DNA extracts from lesionai skin of pat
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Cell Trafficking Networks in Cutaneous T Cell Lymphomaoma (CTCL). To further characterize these cells and to study the role that activation signals and adhesion molecules play in the cellular dynamics of CTCL, immunohistochemical staining of formalin fixed, paraffin embedded tissue sections was used to study expression of high endothelial cell antigen-
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