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Titlebook: Bacteria and Complement; Michael Loos Book 1985 The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer-Verla

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期刊全称Bacteria and Complement
影响因子2023Michael Loos
视频video
学科分类Current Topics in Microbiology and Immunology
图书封面Titlebook: Bacteria and Complement;  Michael Loos Book 1985 The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer-Verla
影响因子Deficiencies in any of the defense mechanisms of the host can lead to severe microbial infections; these are of clinical relevance. Broad up-to-date knowledge in this field allows identification of many unspecific as well as highly specific defense reactions involved in the struggle against infectious diseases. On the other hand, protective structures on the microbial cell surfaces have been adapted and improve the counterpart‘s chances of survival. In particular, it has been considered that the great diversity of the bacterial envelopes not only determines the anatomical location of the tissue injury but also induces activation of distinct parts of the complex defense system. The specific defense mechanism, whose most prominent constituent is provided by the antibo
Pindex Book 1985
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Mathematics and Its Applicationsclassical” pathway:. C1q, C1r, C1s, C4, C2, C3, C5, C6, C7, C8, and C9. There are three proteins of the “alternative” pathway (IUIS-WHO Nomenclature Committee 1981) B, D, and P. Finally, there are four control proteins: C1 inhibitor (Cl¯ INH) and C4b binding protein (C4b-bp) for the classical pathwa
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Gary G. Schwartz,Leonard A. Rosenblumffect is abolished when serum has been incubated for 30 min at 56° C. Gram-positive bacteria are less sensitive than gram-negative bacteria to direct killing, although gram-positive cocci are opsonized by the action of serum mediated by antibodies and complement (. et al. 1968; . et al. 1969). Norma
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Development of Stability Indicating Methodst defence against invading microorganisms and in cooperation with cellular and humoral immune mechanisms. Pathogenic bacteria can be divided into two groups with regard to their fate within phagocytes: (a) extracellular bacteria which are promptly killed after phagocytosis and (b) facultative intrac
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https://doi.org/10.1007/978-0-387-85627-8 that fresh blood could kill certain types of bacteria (. 1985) has evolved detailed understanding of (a) the gram-negative cell wall constituents that activate complement, (b) the role of antibody in initiating or facilitating the process of complement activation, and (c) the biochemical localizati
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