Titlebook: Attempts to Understand Metastasis Formation I; Metastasis-Related M Ursula Günthert,Walter Birchmeier Book 1996 The Editor(s) (if applicabl

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Arnd Hartmanns,Bram Kohlen,Peter Lammichplex control mechanisms, is required in malignancy. The functional role of the enzymatic degradation of ECM by tumor cells has been thought to be the dissolution of occluding matrix, thus enabling the cells to invade into the interstitial matrix of the mesenchyme at the site of a primary tumor, to c

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Soaibuzzaman,Jan Oliver Ringert lungs has often been attributed solely to mechanical entrapment of tumor cell emboli. However, this theory does not explain the early observation that certain. tumor cell types prefer to metastasize to specific target organs. This behaviour was first noticed by Stephen . in 1889. He observed that b

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Dalay Almeida,Florian Jamain,Thierry Lecomteoteins that are capable of binding to laminin (MERCURIO1990). Laminin receptors include several integrins (ABELDAand BUCK 1990), as well as a variety of nonintegrin proteins (MERCURIOI 990), including a 67-kDa high-affinity laminin receptor.

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Eduard Baranov,Axel Legay,Martin Vivianthe latter group including CD44 (. and . 1988; . et al. 1988; . and . 1990; . and . 1990; . 1990; . 1990; . and . 1992; . and .-. 1993; . et al. 1993; . and . 1994; . and . 1994;). Especially integrins, the lg superfamily, and HCAM consist of a multitude of members, a number which is increased by co

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crumble

Plasminogen Activators and Angiogenesis,ar ECM and endothelial cell invasion into the tissue to be vascularized. A number of studies have shown that, among the proteinases involved in angiogenesis, components of the plasminogen activator (PA)-plasmin system play a central role in endothelial cell migration and invasion.

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Different Roles for Plasminogen Activators and Metalloproteinases in Melanoma Metastasis,plex control mechanisms, is required in malignancy. The functional role of the enzymatic degradation of ECM by tumor cells has been thought to be the dissolution of occluding matrix, thus enabling the cells to invade into the interstitial matrix of the mesenchyme at the site of a primary tumor, to c

corpuscle

Lu-ECAM-1 and DPP IV in Lung Metastasis, lungs has often been attributed solely to mechanical entrapment of tumor cell emboli. However, this theory does not explain the early observation that certain. tumor cell types prefer to metastasize to specific target organs. This behaviour was first noticed by Stephen . in 1889. He observed that b

verdict

Model Systems of Carcinoma Cell Dispersion,ncluding colon and breast. Whether or not these phenotypic alterations can be regarded as authentic epithelium-to-mesenchyme transitions (EMTs), similar to those observed during embryogenesis, remains to be clarified. Nevertheless, since the maintenance of epithelial cell differentiation acts as a s