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Titlebook: Atlas of Normal Imaging Variations of the Brain, Skull, and Craniocervical Vasculature; Alexander M. McKinney Book 2017 Springer Internati

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发表于 2025-3-21 17:47:33 | 显示全部楼层 |阅读模式
期刊全称Atlas of Normal Imaging Variations of the Brain, Skull, and Craniocervical Vasculature
影响因子2023Alexander M. McKinney
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发行地址Presents normal imaging variations of the brain, skull, and craniocervical vasculature.Offers a spectrum of appearances for each variant with accompanying 3D imaging.Explores common artifacts on MR an
图书封面Titlebook: Atlas of Normal Imaging Variations of the Brain, Skull, and Craniocervical Vasculature;  Alexander M. McKinney Book 2017 Springer Internati
影响因子This atlas presents normal imaging variations of the brain, skull, and craniocervical vasculature. Magnetic resonance (MR) imaging and computed tomography (CT) have advanced dramatically in the past 10 years, particularly in regard to new techniques and 3D imaging. One of the major problems experienced by radiologists and clinicians is the interpretation of normal variants as compared with the abnormalities that the variants mimic. Through an extensive collection of images, this book offers a spectrum of appearances for each variant with accompanying 3D imaging for confirmation; explores common artifacts on MR and CT that simulate disease; discusses each variant in terms of the relevant anatomy; and presents comparison cases for the purpose of distinguishing normal findings from abnormalities. It includes both common variants as well as newly identified variants that are visualized by recently developed techniques such as diffusion-weighted imaging and multidetector/multislice CT. Thebook also highlights normal imaging variants in pediatric cases. .. ..Atlas of Normal Imaging Variations of the Brain, Skull, and Craniocervical Vasculature. is a valuable resource for neuroradiologist
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Cerebellar Tonsillar Ectopiaas the degree of descent/position (in millimeters) of the tonsils has a normal distribution relative to age. Traditionally defined, a Chiari 1 malformation was simply defined as a hindbrain and skull anomaly consisting of “tonsillar herniation” below the foramen magnum; however, it was later defined
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Cerebellar Flocculus Pseudomasslly on noncontrast MRI sequences. Since the flocculus juts out into the CPA and is surrounded by cerebrospinal fluid, asymmetry (from side to side), volume averaging (with normal adjacent cerebellar hemispheres or the middle cerebellar peduncle), and beam-hardening artifact (from adjacent quite dens
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Cranial Nerve VII: Normal Contrast Enhancement on Magnetic Resonance Imagingl auditory canal (IAC) are quite common. Such MRI examinations typically employ postcontrast T1WI with fat suppression in order to augment visualization of an abnormally enhancing CN7 (e.g., Bell palsy, Ramsay Hunt syndrome, Lyme disease, leukemia, sarcoidosis) or CN8 (e.g., schwannoma, labyrinthiti
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Cavernous Sinus Fat and Pseudomasses(CT) and may simulate air or gas. Without proper windowing the hypodensity on CT can lead to undue concern for air emboli or gas from a fracture involving the skull base (such as gas from the sphenoid sinus or petrous apices), for an arachnoid cyst, or it may even be windowed appropriately but consi
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Pituitary Variations, Artifacts, Primary Empty Sella, and Incidentalomas) and the posterior lobe (neurohypophysis). The adenohypophysis arises from the Rathke pouch, which represents an upward invagination of the oral ectoderm; the adenohypophysis is also called the anterior lobe and secretes a number of important endocrine hormones (under direction from the hypothalamu
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Liliequist Membrane, particularly during endoscopic third ventriculostomy. This membrane is generally located posterior to the pituitary infundibulum (stalk), anterior to the interpeduncular cistern of the midbrain, inferior to the third ventricular floor, superior to the prepontine cistern, and medial to the tip of t
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Pineal Gland: Normal Size, Appearance, and Enhancementn weight and volume with age and can even gradually increase in adulthood. Also, the degree of calcification of the pineal gland should not significantly increase after 30–40 years of age. The glands are typically not calcified in children younger than 5 years of age but may progressively calcify in
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