Titlebook: Atlas of Male Reproductive Pathology; I. D. Ansell Book 1985 I. D. Ansell 1985 biopsy.carcinoma.cell.embryo.testis.tumor

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女歌星

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-Optimal Designs for Exponential Models,rtion of the prostate. The central location of these tumours would also suggest that they might be more suitable for radical surgery than the typical peripheral cancer but this has yet to be adequately evaluated..

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,Testis — Developmental Abnormalities,session of an XY set of sex chromosomes induces the indifferent gonads to develop into testes which then secondarily induce the formation of the male genital tract and external genitalia from their indifferent primordia.

Inoperable

,Testis — Inflammatory Conditions,en in the investigation of male infertility (. Chapter 3). Syphilis of the testis is now a clinical rarity in the UK, so that the characteristic plasma cell infiltrate, interstitial fibrosis, endarteritis and gumma formation (Figure 9.1) is now only seen in Britain in the slide collections of older colleagues or in old student teaching sets!

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escalate

,Penis, Urethra and Scrotum — Benign Conditions,r they are rarely the site of suppuration or indeed of any significant pathology and will not be further discussed. Along the epithelium of the penile urethra there are foci of columnar cells and these may lead into the periurethral glands of Littré (Figure 16.2) which lie within the erectile tissue of the corpus spongiosum.

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, photographs of macro­ scopic specimens are also used to illustrate important features in distinguishing different pathological con­ ditions. Special emphasis is placed on the small biopsy specimens obtained from prostate and testis in modern urological practice and the importance of clinico­ patho

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https://doi.org/10.1007/0-387-31610-8mnar ciliated cells containing much brown lipofuscin pigment are interspersed with smaller cells containing pale secretions (Figure 10.1). The cells lining the ductus epididymis (and the vas deferens also) are also columnar but have little or no intracellular pigment and have prominent apical stereocilia (Figure 10.2).