Titlebook: Atlas of HEART FAILURE; Cardiac Function and Wilson S. Colucci (Professor of Medicine, Chief) Book 2004Latest edition Springer Science+Busi

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yndrome. Designed to provide a detailed and comprehensive visual exposition of all aspects of cardiac function and dysfunction, this atlas contains several hundred images, each accompanied by detailed captions, carefully selected by expert authors, and reviewed by the editor.978-1-4757-4558-0

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Molecular and Cellular Events in Myocardial Hypertrophy and Failure,ers, and peptide growth factors. Genetic manipulations in small animal models are refining our understanding of the stimuli and molecular events that lead to progression of heart failure. Although much remains to be learned about how these stimuli interact with signaling pathways to regulate the rem

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Inhibition of the Renin-Angiotensin-Aldosterone System,ent arteriole [2]. Renin system components have been identified in the myocardium and vasculature, where they adversely affect fibrosis and remodeling, as well as cellular dysfunction. These findings suggest that the renin-angiotensinaldosterone system has effects on cardiac function beyond altering

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https://doi.org/10.1007/978-3-642-67575-1ers, and peptide growth factors. Genetic manipulations in small animal models are refining our understanding of the stimuli and molecular events that lead to progression of heart failure. Although much remains to be learned about how these stimuli interact with signaling pathways to regulate the rem

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S. M. Larson,J. S. Rasey,D. R. Allenhamber volume > mass) or a concentric (chamber mass > volume) pattern of ventricular growth. Although remodeling in response to a pathologic condition can in one sense be considered adaptive because it permits the restoration of pump function in the face of an imposed hyperfunctional condition, the

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Frontiers in Numerical Analysisent arteriole [2]. Renin system components have been identified in the myocardium and vasculature, where they adversely affect fibrosis and remodeling, as well as cellular dysfunction. These findings suggest that the renin-angiotensinaldosterone system has effects on cardiac function beyond altering