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Titlebook: Atherosclerosis and Cardiovascular Disease; 7th International Me Giancarlo Descovich,Antonio Gaddi,Sergio Lenzi Book 1990 Kluwer Academic P

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those not directly involved to keep up with the latest developments. The papers presented at the 7th Meeting on Atherosclerosis and Cardiovascular Disease, held in Bologna, as part of the Ninth Centenary of the foundation of the University of Bologna, have been collected together here with the aim
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The sympatho-vagal balance and arterial hypertensionlterations in neural regulatory activities could be one of these factors (2–4), if not the prevailing one, at least in the early phases of this disorder of regulation. Along this line of reasoning, attention was focused on the sympathetic tone and on the direct or indirect signs of its possible enhanced level (2–4).
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FH gene phenotypic expression: insight for therapeutic strategy of xanthomata and of xanthelasmas. The aim of this paper is to discuss the current diagnostic possibilities in relation to the clinical-laboratory picture and to the therapeutic needs and cardiovascular instrumental diagnosis of the FH patient.
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The effect of coronary heart disease prevention on the prevention of non-cardiovascular diseasest some of the other, major chronic diseases as well [1]. Of the latter, the most important is cancer in its different manifestations. During the last few years, a high level of consensus has been reached that CHD is, indeed, preventable through risk factor control and that both a population and a hi
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The meaning and relevance of intervention trials in preventioning already established risk factors. In the case of atherosclerotic diseases only the third, least satisfactory approach is amenable to test the hypothesis of preventability by means of intervention trials. Prevention through correction of already established high risk is a fringe benefit of the pu
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Prospects for primary prevention of coronary heart diseaseent categories including: atherogenic personal attributes, living habits which promote them, signs of a compromised circulation and host susceptibility to these risk factors. Modifiable atherogenic risk attributes include blood lipids, blood pressure, glucose tolerance and fibrinogen. The risk assoc
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