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Titlebook: Artificial Liver Support; Concepts, Methods, R G. Brunner,M. Mito Book 1992Latest edition Springer-Varlag Berlin Heidelberg 1992 Hepatologi

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期刊全称Artificial Liver Support
期刊简称Concepts, Methods, R
影响因子2023G. Brunner,M. Mito
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图书封面Titlebook: Artificial Liver Support; Concepts, Methods, R G. Brunner,M. Mito Book 1992Latest edition Springer-Varlag Berlin Heidelberg 1992 Hepatologi
影响因子Continuing progress has been made since the first edition of Artificial Liver Support was published. Liver transplantation has however become an estab­ lished therapy for a relatively small number of patients who remain patients for life. There therefore continues to be a great need for the development of other forms of artificial liver support. Improved intensive care utilizing improved plasma exchange, dialysis, sclerotherapy, and intracranial pressure monitoring have improved survival in fulminant hepatic failure. Progress has also been made in lipid membrane detoxification, in cell cultures, and in cell transplantation, and the isolation of various liver cell growth factors has led to deep insight into the mechanisms of liver regeneration. This book gives the clinician and the researcher detailed information about established new methods of clinic work and laboratory research, and describes new experimental approaches indicating the direction of future research. G. BRUNNER M. Mno Preface to the First Edition The regenerative capacity of the liver cell is almost unlimited. Therefore after acute liver damage, be it viral, toxic, hypoxic, or surgical in origin, restitutio ad integ
Pindex Book 1992Latest edition
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https://doi.org/10.1007/978-3-319-13829-9f these regulatory mechanisms is the compensatory growth of the liver (liver regeneration) that occurs after partial hepatectomy in rats or mice. An almost exact human counterpart for this experimental system now exists, as shown by studies of the regulation of the size and growth of transplanted hu
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https://doi.org/10.1007/978-3-030-44692-5e subjected to daily partial hepatectomy by a bird, after he stole the secret of fire from the gods and passed it to the humans. Despite many years of study of liver regeneration, the detailed nature of the controlling factors which trigger or modulate this phenomenon has only recently started to be
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Afterword: Making a Noise About the Silence,may in some cases be associated with massive necrosis of liver cells; in other circumstances, metabolic abnormalities, often accompanied morphologically by microvesicular steatosis, result in a similar syndrome despite the absence of significant necrosis. Except when the syndrome evolves during the
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Afterword: Making a Noise About the Silence,utcome may also be due to lack of adequate treatment of treatable complications, such as gastrointestinal hemorrhage, or to complications due] to interventions, such as septicemia from intravenous lines. This makes monitoring of liver function essential. Furthermore, monitoring of the factors determ
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https://doi.org/10.1057/9780230372580ich it can remove include bilirubin, bile acid, and endotoxin. Of these, endotoxin (ET) is a constituent of the cell wall of gram-negative bacilli, which under normal conditions may be absorbed from the gastrointestinal tract into the portal blood and be removed by the reticuloendothelial cells of t
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