Titlebook: Arteriosclerosis; New Insights into Pa H. Just,W. Hort,A. M. Zeiher Conference proceedings 1994 Dr. Dietrich Steinkopff Verlag GmbH & Co. K

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Therapeutic approaches to the control of coronary atherosclerosis, lipid, monocyte adhesion followed by penetration across the endothelium, the conversion of monocytes to macrophages and smooth muscle cell proliferation and migration are some of the events involved in the early stages of lesion formation. Late events include the formation of excess ground substanc

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Role of calcium antagonists in progression of arteriosclerosis. Evidence from animal experiments an arteries during the whole life span (0–90 years). However, this normal ratio increasingly changes as soon as arteriosclerotic alterations of the coronary walls set in. Accordingly, the mural calcium content steadily rises from fatty streaks over severe arteriosclerosis and, lastly, seems to reach a

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,Lipid-lowering therapy — Implications for the prevention of atherosclerosis,dies, epidemiologic surveys, and intervention studies in man. There have been recommendations by national and international advisory boards regarding the aims and means of such interventional strategies. These have not been unanimously accepted, however. The present article will review the evidence

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https://doi.org/10.1007/978-3-030-95833-6Adaptive thickening occurs in defined locations from birth and represents a self-limited response of the intima to hemodynamic forces present within specific locations. Adaptive thickening does not indicate or presage an atherosclerotic lesion. However, some of the identical intima locations (progre

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Energy Technologies and Economicsain cellular events: 1) activation of macrophages leading to lipoprotein phagocytosis by scavenger cells, and 2) proliferation of smooth muscle cells (SMC). SMC-like cells producing collagenous fibers and extracellular matrix are particularly involved in the formation of the so-called fibrous caps s