Titlebook: Apoptosis in Carcinogenesis and Chemotherapy; Apoptosis in cancer George G. Chen,Paul B.S. Lai Book 2009 Springer Science+Business Media B.

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书目名称Apoptosis in Carcinogenesis and Chemotherapy影响因子(影响力)




书目名称Apoptosis in Carcinogenesis and Chemotherapy影响因子(影响力)学科排名




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书目名称Apoptosis in Carcinogenesis and Chemotherapy网络公开度学科排名




书目名称Apoptosis in Carcinogenesis and Chemotherapy被引频次




书目名称Apoptosis in Carcinogenesis and Chemotherapy被引频次学科排名




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书目名称Apoptosis in Carcinogenesis and Chemotherapy年度引用学科排名




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Book 2009bringing together an important pool of up-to-date knowledge to light and further our interest in this exciting and expanding ?eld. Arthur K. C. Li Emeritus Professor of Surgery The Chinese University of Hong Kong v Preface The role of apoptosis in cancer development and emerging treatment strategies

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Anti-Cancer Strategy of Transitional Cell Carcinoma of Bladder Based on Induction of Different Typer response. Current strategy is to decipher the complexities of death pathways in order to identify key effector molecules and their function. As their mechanisms have been elucidated, so has the knowledge that aberrations can occur at many points along the pathway, interfering with normal function.

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Apoptosis in Carcinogenesis and Chemoherapy of the Uterine Cervix,d inhibit proliferation by arresting cell cycle progression. Due to its central role in the development of cancer, maintenance of transformed phenotype or response to therapy, E6/E7 oncogenes are unique targets for novel therapeutic strategies. RNA interference has evolved from the recent studies as

bypass

Apoptosis in Cutaneous Melanoma,utcome. APAF-1 loss or downregulation was demonstrated to be an important factor in melanoma’s aggressive behavior. The use of apoptosis related factors as biomarkers can be applied to assess both tumor tissue and blood. By assessing tumors and identifying their potential of resistance to therapeuti

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,Apoptosis in Carcinogenesis and Chemotherapy – Esophageal Cancer,s. In the last decade, it has been demonstrated that resistance to apoptotic stimuli is a hallmark feature of esophageal cancer and a complex network of pro- and anti-apoptotic proteins that governs the tight regulation along apoptotic pathways in regards of esophageal carcinogenesis and chemotherap

perpetual

Apoptosis and the Tumor Microenvironment in Hematologic Malignancies,hich often exhibit upregulation of anti-apoptotic pathways, are typically the cause of relapse of hematologic diseases. Thus, targeting these tumor cells along with the tumor microenvironment in which the tumor cells reside is vital in overcoming the devastating effects associated with hematologic m

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Elemente einer analytischen Hydrologied inhibit proliferation by arresting cell cycle progression. Due to its central role in the development of cancer, maintenance of transformed phenotype or response to therapy, E6/E7 oncogenes are unique targets for novel therapeutic strategies. RNA interference has evolved from the recent studies as