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Titlebook: Antithrombotics; Andrew C. G. Uprichard (Vice President),Kim P. Gal Book 1999 Springer-Verlag Berlin Heidelberg 1999 Antithrombin.Arzneimi

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Roundtable on Numerical Methods, Ilb/IIIa inhibitor). Given the novelty of abciximab, a substantial portion of this chapter will review the preclinical and clinical development of this monoclonal antibody as antiplatelet therapy for the treatment of ischemic complications associated with percutaneous coronary revascularization. A
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https://doi.org/10.1007/978-3-642-14968-9n and treatment of venous thromboembolism. The low molecular weight heparins have also been used in clinical trials for the prevention and treatment of arterial thrombosis. In this chapter, we review the problems related to the use of unfractioned heparin, compare the low molecular weight heparins w
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Solitons and Charge Density Waves,o activate factors V, VIII and XI. Recruitment of these additional factors begins the “augmentation stage” which can provide large amounts of additional thrombin that plays a dominant role in subsequent thrombus formation. In both the classical coagulation cascade and the more current tissue factor
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Overview of Radiation from Dense Media, of additional thrombin and, thereby, fibrin. Although the TF/VIIa complex is not directly responsible for the production of all of the thrombin needed to form a complete thrombus, it plays the crucial role of initiating the process.
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R. H. Avakian,K. A. Ispirian,V. J. Yaralov exposure of negatively charged phospholipids. These clotting reactions are offset by natural anticoagulant mechanisms, the most important of which are thought to include the tissue factor pathway inhibitor (Chap. 15), antithrombin-heparin (Chap. 9) and the protein C anticoagulant pathway (depicted
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Monitoring Antithrombotic Therapy,te the antithrombotic effects from the global anticoagulant effects of currently available agents, the therapeutic window of such agents tends to be narrow, and dose adjustment requires regular therapeutic monitoring.
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