Titlebook: Animal Models; Disorders of Eating John B. Owen,Janet L. Treasure,David A. Collier Book 2001 Springer Science+Business Media Dordrecht 200

Congruous

迅速飞过

The Halothane Gene, Leanness and Stress Susceptibility in Pigsercising and hence improved muscling and reduced fat in pigs. This explanation for the associations between .. and reduced fat is currently more plausible than the alternative explanation of a fat/lean gene very closely linked to the . locus.

obeisance

轻信

relate these to similar conditions in humans. With advances in understanding the nature of these disorders and their biological basis, it seems timely to assess what cross-species comparisons can tell us about the general underlying factors at work. This may also help to delineate what may be a gen

痛苦一生

Economic Analysis of Music Copyrightal. However, there are several interesting alterations of the distribution of many appetite regulating peptides in the hypothalamus of mutant mice. Serum leptin levels are also altered in . mice. The . mouse is a useful model of malnutrition and particularly for exploring the role of hypothalamic regulation of food intake.

Flu表流动

inchoate

Collar

https://doi.org/10.1007/978-94-009-2667-7dromes are presented. These comparisons illustrate the important cross-species effects of certain genes, point out potential candidate genes with as yet undemonstrated roles in human obesity, and highlight possible physiological pathways for further investigation in human obesity studies.

厌恶

https://doi.org/10.1007/978-94-011-6483-2ue to its complex etiology involving behavior, energy expenditure, and genetics. In this chapter, leptin and its involvement in the genetics and physiology of obesity will be discussed, including its therapeutic potential in the clinical treatment of this health condition.

音乐学者

Obesity Syndromesdromes are presented. These comparisons illustrate the important cross-species effects of certain genes, point out potential candidate genes with as yet undemonstrated roles in human obesity, and highlight possible physiological pathways for further investigation in human obesity studies.