Titlebook: Animal Models of Allergic Disease; Methods and Protocol Kumi Nagamoto-Combs Book 2021 Springer Science+Business Media, LLC, part of Springe

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P. M. Mohan,P. Dhivya,K. Narayanamurthye first mouse model of inhalation-driven peanut allergy. We administered PN flour intranasally to naïve wild-type mice twice a week for 4 weeks, followed by intraperitoneal challenge with PN extract. Exposure of mice to PN flour sensitized them without addition of adjuvants, and mice developed PN-sp

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Rafael Reyna-Hurtado,Khiavett Sanchez-Pinzóntization. However, the molecular mechanisms involved in the disease etiology are not completely understood. A complex cellular and tissue network linking to food allergy makes it difficult to understand the molecular mechanism of allergenicity. Animal models are valuable tools to deduce basic princi

Cholagogue

Allodynia

Salvador Mandujano,Rafael Reyna-Hurtadorevalence of chronic conditions is on the rise, milk allergy is a growing public health concern. To elucidate underlying mechanisms and develop therapeutic strategies, reliable animal models are essential research tools. Sensitization to a milk protein is the principal procedure for establishing ani

Insulin

Implementation of Monitoring Systemsrmalities are thought to contribute to the development of AD, the precise pathogenic mechanism remains to be elucidated. We have developed a unique, diet-induced AD mouse model based on the findings that deficiencies of certain polyunsaturated fatty acids and starches cause AD-like symptoms in hairl

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Implementation of Monitoring Systemsy the exposure to haptens that leads to an initial phase known as sensitization. During this phase, hapten–protein complexes presented by antigen-presenting cells activate a T-cell-mediated response, leading to the generation of memory cells against the hapten. Upon re-exposure to the same hapten, t

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Regulation of Environmental Qualityonsiveness, and obstruction. Here, we present the allergic asthma models, in which mice are hypersensitized to ovalbumin (OVA) and house dust mite (HDM). These models mimic the major characteristics of human asthma including the eosinophilic inflammation and hyperactivity of the airway, overproducti

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Regulation of Environmental Qualityitis to study immunological aspects of this condition. In this chapter, we will describe ragweed-pollen-induced experimental allergic conjunctivitis (mostly driven by adaptive immunity), and papain-soaked contact lens-induced experimental allergic conjunctivitis (mostly driven by innate immunity). G

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