Titlebook: Animal Models in Parasitology; A Symposium held at Dawn G. Owen Textbook 1982Latest edition The contributors 1982 model.parasite.parasitol

REIGN

Preamble

Social Paradoxes of Growth and Civil Economye unravelling of the immune mechanisms which, in normal animals, are responsible for the maintenance of an equable host-parasite relationship. Such studies on intestinal protozoa are comparatively sparse, but this chapter attempts to consider the available literature, mainly with respect to infectio

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Giangiacomo Nardozzi,Marcello de Cecco completely athymic (Loor and Roelants, 1974) but may contain a few functional T cells (Rudnicka ., 1978). Their usefulness, in a field where strain differences in parasite sensitivity are very marked, has recently been greatly enhanced by the transfer of the ./. genotype into several well-known inb

Anthem

Economic Theory and Social Justicenearly all immunological stimuli (Miller, 1961; Good ., 1962; Jankovic ., 1962). Since that time T-cell-deprived animals, rendered so by neonatal thymectomy (as in the earliest experiments), or more conveniently by adult thymectomy, and total body irradiation (Miller ., 1963) or by congenital thymic

modifier

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Price Determination for a Single Project macrophage as an effector cell within the immune system of the host, on the one hand, and all the other cells which can act as a host to parasitic protozoa ., on the other hand. The latter is perhaps unfortunate, as it relegates from consideration here such host cell and obligate intracellular para

Mnemonics

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Genetic Manipulation of the Host as a Method in ParasitologyThese defined animals include inbred, congenic and recombinant inbred strains, F. hybrids and a wide range of different mutations. Selection for resistance and susceptibility to the parasite or for immune function is another promising method both of developing better hosts and of studying the host-p

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Adornment

Mouse Models of Genetic Variation in Resistance to Helminth Parasiteson in ability to develop resistance to helminth infections (Wakelin, 1978a). The existence of variation has been inferred from differences in the prevalence and intensity of infection, as well as from differences in pathological manifestations in the host. By their nature, such observations have bee