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Titlebook: Anesthesia and the Lung; T. H. Stanley,R. J. Sperry Book 1989 Kluwer Academic Publishers 1989 COPD.hemodynamics.hypoxia.intubation.pathoph

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发表于 2025-3-21 17:30:54 | 显示全部楼层 |阅读模式
期刊全称Anesthesia and the Lung
影响因子2023T. H. Stanley,R. J. Sperry
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学科分类Developments in Critical Care Medicine and Anaesthesiology
图书封面Titlebook: Anesthesia and the Lung;  T. H. Stanley,R. J. Sperry Book 1989 Kluwer Academic Publishers 1989 COPD.hemodynamics.hypoxia.intubation.pathoph
影响因子Theodore H. Stanley, M. D. Anesthesia and the Lung contains the Refresher Course manuscripts of the presentations of the 34th Annual Postgraduate Course in Anesthesiology which took place at The Cliff Conference Center in Snowbird, Utah, February 17-21, 1989. The chapters reflect recent advances in the diagnosis, pre-, intra-, and postoperative anesthetic management of patients with lung disease, presenting for pulmonary and non-pulmonary surgery. They also deal with ventilation-perfusion issues, the lung as a metabolic organ, the effects of anesthesia on pulmonary mechanics and pulmonary blood flow. In addition there are chapters that will focus around hypoxia; regional differences in the lung; pulmonary surfactant; recent advances in the understanding of pulmonary edema; high altitude disease; anesthesia and the control of breathing; recent developments in oximetry; instrumentation designed to measure pulmonary oxygen tension, P0 and PC0 trans­ 2 2 cutaneously; differential lung ventilation; reactive airways; septic shock; the adult respiratory distress syndrome and numerous aspects of ventilatory support. The purposes of the textbook are to 1) act as a reference for the anesthes
Pindex Book 1989
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Pathophysiology of Pulmonary Edema: Implications for Clinical Management, is called the Starling equation. A review of the history of this equation makes understanding much easier. Ernest Starling (1866–1927) was a prolific investigator who made major contributions in the areas of cardiovascular, endocrine, gut, nutritional, and microvascular physiology.His work on the f
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Control of Breathing,- act to produce one of their more notorious side effects, depression of breathing. I wi11 review dose-response relationships, discuss clinical implications, and explore some of the questions of basic ventilatory control revealed by the way in which these drugs modify breathing.
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Pulmonary Surfactant: An Endogenous Mediator of Alveolar Stability and a Therapeutic Agent,ulates alveolar surface tension, decreasing it at end expiration (preventing closure of the alveoli) and allowing it to increase at end inspiration (contributing to elastic recoil). The discovery that surfactant deficiency is the primary defect in Infant Respiratory Distress Syndrome, and that surfa
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Preparing the Patient With COPD for Surgery,onic bronchitis, obstructive emphysema, asthma, bronchiectasis, mucoviscidosis (cystic fibrosis), and central airway obstruction. Chronic bronchitis is defined clinically as sputum production on most days of the week for at least three months of the year in two or more consecutive years. Pathologica
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Ventilation-Perfusion Mismatching during Anesthesia,finition and of direct relevance to therapy. It presents the lung as though it comprises three compartments — ideally perfused and ventilated alveoli, alveolar dead space and shunt (Figure 1). Physiological dead space is easily quantified by the Bohr equation, and shunt by the shunt equation.
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Oximetry I: Nomenclature, History, Accuracy and Uses,d by exposing the blood to a bubble of O., measuring the O. content by a physical or electrochemical method, and subtracting the dissolved O. determined by measuring Po. after bubble equilibration. O. capacity is not determined by 1.35 times hemoglobin concentration. HbCO and HbMet reduce O. content
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