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Titlebook: Alcohol and Aldehyde Metabolizing Systems-IV; Ronald G. Thurman Book 1980 The Editor(s) (if applicable) and The Author(s), under exclusive

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Genetically Mediated Responses of Microsomal Ethanol Oxidation in Mice5 fold) in both LS and SS mice by 16 days of treatment. Chronic ethanol treatment resulted in a significant induction of ethylmorphine and benzphetamine N-demethylase activity in both LS and SS mice with the SS selected line showing a greater (P<.05) maximal induction. These data are suggestive that
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The Nature and Function of Aldehyde Reductases from Rat Braints a substantial preference for 2-hydroxy aldehydes (Turner and Tipton, 1972b; Wermuth and Münch, 1979). Rat brain and other tissues contain at least one other aldehyde reductase (AR2 or “low-K.”) that is similar to or identical with aldose reductase (EC 1.1.1.21) (Turner and Tipton, 1972b). This la
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The Esa Satellite Communications Controllered from compositional comparisons. A UEP of 12 is approximately half that of lactate dehydrogenase and shows that aldehyde reductase is evolving at twice the rate of glycolytic enzymes. This may indicate a relatively non-essential metabolic role for the enzyme.
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James Reinders,Ben Ashbaugh,Xinmin Tian5 fold) in both LS and SS mice by 16 days of treatment. Chronic ethanol treatment resulted in a significant induction of ethylmorphine and benzphetamine N-demethylase activity in both LS and SS mice with the SS selected line showing a greater (P<.05) maximal induction. These data are suggestive that
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Scheduling Kernels and Data Movement,on of aspartate (5 mM) partially reversed the inhibition of mixed-function oxidation by ethanol. These data indicate that changes in the oxidation-reduction state of pyridine nucleotides due to the metabolism of ethanol and acetaldehyde inhibit the transfer of mitochondrial reducing equivalents into
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