Titlebook: Aging and Cell Structure; Volume 1 John E. Johnson Book 1981 Plenum Press, New York 1981 RNA.aging.biology.brain.cell.cells.complication.me

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Ultrastructure of the Aging Kidney,ted with a decline of glomerular filtration rate and various types of tubular dysfunction (Friedman ., 1972; Korenchevsky, 1961; Epstein, 1979; Papper, 1973). Although gross, anatomic, light microscopic, and functional studies in the aging kidney may be few, they are plentiful in relationship to ultrastructural studies of the kidney in aging man.

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nt concerns of mankind. Even restricting time to the past decade or so and approaching only the biomedical sciences, one still encounters a flood of information in this relatively young research area. The­ ories and ideas abound as though each researcher provides one of his own. This might well be e

evasive

Ordinary Differentiation Equations,pond to primary decay due to biological aging per se. Such changes involve both somatic and autonomic neurons, and transcend at the level of the neuronal cyton, the axon, the Schwann cell, and the motor and sensory nerve endings.

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Herbert F. Jelinek,David J. Cornforth accumulation of pathological lesions but a fundamental process. He further comments on the importance of unifying biological concepts, such as the cell theory and, more recently, the electron microscopic demonstration of the basic similarity of cellular organelles.

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The Mammalian Peripheral Nervous System in Old Age,pond to primary decay due to biological aging per se. Such changes involve both somatic and autonomic neurons, and transcend at the level of the neuronal cyton, the axon, the Schwann cell, and the motor and sensory nerve endings.

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Insect vs. Mammalian Aging, accumulation of pathological lesions but a fundamental process. He further comments on the importance of unifying biological concepts, such as the cell theory and, more recently, the electron microscopic demonstration of the basic similarity of cellular organelles.

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Central Nervous System,ons and neuroglia, but we believe that sufficient information will be given in this account for the reader to use it as a means of gaining access to studies that provide additional information and other views.

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Neurofibrillary and Synaptic Pathology in the Aged Brain,ctious stress. At the same time, genetically programmed time-associated changes in aging brain cells may increase their susceptibility to harmful environmental effects (hormonal, infectious, immunological) and lead to various pathological changes found in the aged brain.